[32P]ATP inhibits the growth of xenografted tumors in nude mice

Cell Cycle. 2012 May 15;11(10):1878-82. doi: 10.4161/cc.19955. Epub 2012 May 15.

Abstract

The search for new therapeutic agents that are effective against cancer has been difficult and expensive. The activity of anticancer candidate agents against human cancer-derived cell lines in immunocompromised mice is an important tool in this search. Because ATP is a naturally occurring small molecule, its radiolabeled form poses many advantages as a potential anticancer therapeutic agent. We previously found that a single, low-dose intravenous injection of [ ( 32) P]ATP inhibited the growth of xenografted tumors in nude mice for up to several weeks. The current study describes the biodistribution and the results and advantages of multi-dose administration of this potential drug. Future studies should investigate the mechanism involved in the possible use of [ ( 32) P]ATP as a cytotoxic agent that homes naturally to the tumor microenvironment.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Adenosine Triphosphate / pharmacokinetics*
  • Adenosine Triphosphate / therapeutic use
  • Animals
  • Antineoplastic Agents / pharmacokinetics*
  • Antineoplastic Agents / therapeutic use
  • HeLa Cells
  • Humans
  • Immunocompromised Host
  • Mice
  • Mice, Nude
  • Neoplasms / drug therapy*
  • Neoplasms / pathology
  • Phosphorus Radioisotopes / chemistry
  • Tissue Distribution
  • Transplantation, Heterologous

Substances

  • Antineoplastic Agents
  • Phosphorus Radioisotopes
  • Adenosine Triphosphate