Pulmonary function reductions among potentially susceptible subgroups of agricultural workers in Colorado and Nebraska

J Occup Environ Med. 2012 May;54(5):632-41. doi: 10.1097/JOM.0b013e31824d2e1c.

Abstract

Objective: Organic dust inhalation has been associated with adverse respiratory responses among agricultural workers. We evaluated factors that may confer increased susceptibility to these health effects.

Methods: We quantified personal work shift exposures to inhalable dust, endotoxin, and its 3-hydroxy fatty acid constituents, and evaluated changes in pulmonary function among 137 grain elevator, cattle feedlot, dairy, and corn farm workers.

Results: Increased dust exposure was associated with work shift reductions in lung function. Although interpretation is limited because of small samples, a suggestion of stronger exposure-response relationships was observed among smokers, as well as workers reporting pesticide/herbicide application, asthma, or allergies, and those with genetic polymorphisms (TLR4) (Pinteraction ≤ 0.05).

Conclusions: A better understanding of factors leading to increased susceptibility of adverse respiratory outcomes is needed to optimize exposure reduction strategies and develop more comprehensive wellness programs.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adolescent
  • Adult
  • Aged
  • Agriculture*
  • Animals
  • Asthma / physiopathology
  • Cattle
  • Colorado
  • Dust
  • Endotoxins / toxicity*
  • Fatty Acids / toxicity*
  • Forced Expiratory Volume / genetics
  • Humans
  • Hypersensitivity / physiopathology
  • Inhalation Exposure / adverse effects*
  • Lung / physiopathology*
  • Male
  • Middle Aged
  • Nebraska
  • Occupational Exposure / adverse effects*
  • Pesticides / toxicity
  • Polymorphism, Genetic
  • Smoking / adverse effects
  • Time Factors
  • Toll-Like Receptor 4 / genetics
  • Vital Capacity / genetics
  • Young Adult

Substances

  • Dust
  • Endotoxins
  • Fatty Acids
  • Pesticides
  • TLR4 protein, human
  • Toll-Like Receptor 4