High resolution copy number analysis of IRF4 translocation-positive diffuse large B-cell and follicular lymphomas

Genes Chromosomes Cancer. 2013 Feb;52(2):150-5. doi: 10.1002/gcc.22014. Epub 2012 Oct 17.

Abstract

Translocations affecting chromosome subband 6p25.3 containing the IRF4 gene have been recently described as characteristic alterations in a molecularly distinct subset of germinal center B-cell-derived lymphomas. Secondary changes have yet only been described in few of these lymphomas. Here, we performed array-comparative genomic hybridization and molecular inversion probe microarray analyses on DNA from 12 formalin-fixed paraffin-embedded and two fresh-frozen IRF4 translocation-positive lymphomas, which together with the previously published data on nine cases allowed the extension of copy number analyses to a total of 23 of these lymphomas. All except one case carried chromosomal imbalances, most frequently gains in Xq28, 11q22.3-qter, and 7q32.1-qter and losses in 6q13-16.1, 15q14-22.31, and 17p. No recurrent copy-neutral losses of heterozygosity were observed. TP53 point mutations were detected in three of six cases with loss of 17p. Overall this study unravels a recurrent pattern of secondary genetic alterations in IRF4 translocation-positive lymphomas.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Adult
  • Aged
  • Aged, 80 and over
  • Base Sequence
  • Child
  • Child, Preschool
  • Chromosome Aberrations
  • Comparative Genomic Hybridization / methods
  • DNA Copy Number Variations*
  • DNA Mutational Analysis
  • Female
  • Germinal Center / metabolism
  • Germinal Center / pathology
  • Humans
  • In Situ Hybridization, Fluorescence
  • Interferon Regulatory Factors / genetics*
  • Lymphoma, Follicular / genetics*
  • Lymphoma, Follicular / metabolism
  • Lymphoma, Follicular / pathology
  • Lymphoma, Large B-Cell, Diffuse / genetics*
  • Lymphoma, Large B-Cell, Diffuse / pathology
  • Male
  • Middle Aged
  • Point Mutation
  • Translocation, Genetic*
  • Tumor Suppressor Protein p53 / genetics
  • Young Adult

Substances

  • Interferon Regulatory Factors
  • Tumor Suppressor Protein p53
  • interferon regulatory factor-4