The battle between rotavirus and its host for control of the interferon signaling pathway

PLoS Pathog. 2013 Jan;9(1):e1003064. doi: 10.1371/journal.ppat.1003064. Epub 2013 Jan 24.

Abstract

Viral pathogens must overcome innate antiviral responses to replicate successfully in the host organism. Some of the mechanisms viruses use to interfere with antiviral responses in the infected cell include preventing detection of viral components, perturbing the function of transcription factors that initiate antiviral responses, and inhibiting downstream signal transduction. RNA viruses with small genomes and limited coding space often express multifunctional proteins that modulate several aspects of the normal host response to infection. One such virus, rotavirus, is an important pediatric pathogen that causes severe gastroenteritis, leading to ~450,000 deaths globally each year. In this review, we discuss the nature of the innate antiviral responses triggered by rotavirus infection and the viral mechanisms for inhibiting these responses.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, N.I.H., Intramural
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Review

MeSH terms

  • Gastroenteritis / immunology
  • Gastroenteritis / virology
  • Host-Pathogen Interactions / immunology*
  • Humans
  • Immunity, Innate
  • Interferons / immunology*
  • Interferons / metabolism
  • Rotavirus / growth & development
  • Rotavirus / immunology*
  • Rotavirus Infections / immunology*
  • Rotavirus Infections / virology
  • Signal Transduction / immunology

Substances

  • Interferons