Uptake and phosphorylation of phosphatidylinositol by rat liver nuclei. Role of phosphatidylinositol transfer protein

Biochim Biophys Acta. 1990 May 22;1044(2):193-200. doi: 10.1016/0005-2760(90)90303-f.

Abstract

The incorporation of phosphatidyl[2-3H]inositol ([3H]PI) from vesicles or microsomal membranes into rat liver nuclei is greatly stimulated by phosphatidylinositol transfer protein (PI-TP). The nuclei are able to phosphorylate [3H]PI, with the production of phosphatidylinositol 4-phosphate (PIP). Recovery of tritiated inositol trisphosphate, inositol phosphate, glycerophosphoinositol and inositol, suggests that in isolated nuclei a large set of enzymes of the PI cycle is present, similar to the enzymes involved in the plasma membrane PI cycle. Incubation with [gamma-32P]ATP shows that isolated nuclei are able to phosphorylate endogenous PI to PIP and phosphatidylinositol 4,5-bisphosphate (PIP2). In the presence of exogenous PI and detergent the synthesis of PIP is increased, indicating that in nuclei the PI pool is suboptimal for the PI-kinase activity. The present study suggests that PI-TP may be involved in providing substrates for PI metabolism at the nuclear level.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Carrier Proteins / metabolism*
  • Cell Fractionation
  • Cell Nucleus / metabolism*
  • Chromatography, Thin Layer
  • Kinetics
  • Liposomes / metabolism
  • Liver / metabolism*
  • Liver / ultrastructure
  • Male
  • Membrane Proteins*
  • Phosphatidylinositols / metabolism*
  • Phospholipid Transfer Proteins
  • Phosphorylation
  • Rats
  • Rats, Inbred Strains

Substances

  • Carrier Proteins
  • Liposomes
  • Membrane Proteins
  • Phosphatidylinositols
  • Phospholipid Transfer Proteins