Acute stress enhances adult rat hippocampal neurogenesis and activation of newborn neurons via secreted astrocytic FGF2

Elife. 2013 Apr 16:2:e00362. doi: 10.7554/eLife.00362.

Abstract

Stress is a potent modulator of the mammalian brain. The highly conserved stress hormone response influences many brain regions, particularly the hippocampus, a region important for memory function. The effect of acute stress on the unique population of adult neural stem/progenitor cells (NPCs) that resides in the adult hippocampus is unclear. We found that acute stress increased hippocampal cell proliferation and astrocytic fibroblast growth factor 2 (FGF2) expression. The effect of acute stress occurred independent of basolateral amygdala neural input and was mimicked by treating isolated NPCs with conditioned media from corticosterone-treated primary astrocytes. Neutralization of FGF2 revealed that astrocyte-secreted FGF2 mediated stress-hormone-induced NPC proliferation. 2 weeks, but not 2 days, after acute stress, rats also showed enhanced fear extinction memory coincident with enhanced activation of newborn neurons. Our findings suggest a beneficial role for brief stress on the hippocampus and improve understanding of the adaptive capacity of the brain. DOI:http://dx.doi.org/10.7554/eLife.00362.001.

Keywords: FGF2; Rat; astrocytes; hippocampus; neurogenesis; stem cell; stress.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Acute Disease
  • Adaptation, Physiological
  • Adaptation, Psychological
  • Age Factors
  • Animals
  • Animals, Newborn
  • Astrocytes / metabolism*
  • Behavior, Animal
  • Cell Proliferation
  • Corticosterone / pharmacology
  • Culture Media, Conditioned / metabolism
  • Disease Models, Animal
  • Extinction, Psychological
  • Fear
  • Fibroblast Growth Factor 2 / drug effects
  • Fibroblast Growth Factor 2 / genetics
  • Fibroblast Growth Factor 2 / metabolism*
  • Gene Expression Regulation
  • Hippocampus / drug effects
  • Hippocampus / metabolism*
  • Hippocampus / physiopathology
  • Male
  • Memory
  • Neural Stem Cells / metabolism*
  • Neurogenesis* / drug effects
  • Paracrine Communication*
  • RNA, Messenger / metabolism
  • Rats, Sprague-Dawley
  • Signal Transduction
  • Stress, Psychological / genetics
  • Stress, Psychological / metabolism*
  • Stress, Psychological / physiopathology
  • Stress, Psychological / psychology
  • Time Factors

Substances

  • Culture Media, Conditioned
  • RNA, Messenger
  • Fibroblast Growth Factor 2
  • Corticosterone