Immunological profile of HTLV-1-infected patients associated with infectious or autoimmune dermatological disorders

PLoS Negl Trop Dis. 2013 Jul 25;7(7):e2328. doi: 10.1371/journal.pntd.0002328. Print 2013.

Abstract

In the present study, the frequency, the activation and the cytokine and chemokine profile of HTLV-1 carriers with or without dermatological lesions were thoroughly described and compared. The results indicated that HTLV-1-infected patients with dermatological lesions have distinct frequency and activation status when compared to asymptomatic carriers. Alterations in the CD4(+)HLA-DR(+), CD8(+) T cell, macrophage-like and NKT subsets as well as in the serum chemokines CCL5, CXCL8, CXCL9 and CXCL10 were observed in the HTLV-1-infected group with skin lesions. Additionally, HTLV-1 carriers with dermatological skin lesions showed more frequently high proviral load as compared to asymptomatic carriers. The elevated proviral load in HTLV-1 patients with infectious skin lesions correlated significantly with TNF-α/IL-10 ratio, while the same significant correlation was found for the IL-12/IL-10 ratio and the high proviral load in HTLV-1-infected patients with autoimmune skin lesions. All in all, these results suggest a distinct and unique immunological profile in the peripheral blood of HTLV-1-infected patients with skin disorders, and the different nature of skin lesion observed in these patients may be an outcome of a distinct unbalance of the systemic inflammatory response upon HTLV-1 infection.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Autoimmune Diseases / complications*
  • Carrier State / epidemiology
  • Carrier State / immunology
  • Female
  • HTLV-I Infections / complications*
  • HTLV-I Infections / epidemiology
  • HTLV-I Infections / immunology*
  • Human T-lymphotropic virus 1 / immunology*
  • Human T-lymphotropic virus 1 / isolation & purification
  • Humans
  • Killer Cells, Natural / immunology
  • Lymphocyte Subsets / immunology
  • Macrophages / immunology
  • Male
  • Middle Aged
  • Skin Diseases / complications*
  • Viral Load

Grants and funding

ATC and OAMF thank CNPq for fellowships (PQ). ACCA thanks the Programa Nacional de Pós-Doutorado (PNPD) for a fellowship. The authors also thank the program for technological development in tools for health - PDTIS, FAPEMIG, FIOCRUZ and Fundação Hemominas for financial support. The funders of this research work had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.