Partial antiviral activities detection of chicken Mx jointing with neuraminidase gene (NA) against Newcastle disease virus

PLoS One. 2013 Aug 16;8(8):e71688. doi: 10.1371/journal.pone.0071688. eCollection 2013.

Abstract

As an attempt to increase the resistance to Newcastle Disease Virus (NDV) and so further reduction of its risk on the poultry industry. This work aimed to build the eukaryotic gene co-expression plasmid of neuraminidase (NA) gene and myxo-virus resistance (Mx) and detect the gene expression in transfected mouse fibroblasts (NIH-3T3) cells, it is most important to investigate the influence of the recombinant plasmid on the chicken embryonic fibroblasts (CEF) cells. cDNA fragment of NA and mutant Mx gene were derived from pcDNA3.0-NA and pcDNA3.0-Mx plasmid via PCR, respectively, then NA and Mx cDNA fragment were inserted into the multiple cloning sites of pVITRO2 to generate the eukaryotic co-expression plasmid pVITRO2-Mx-NA. The recombinant plasmid was confirmed by restriction endonuclease treatment and sequencing, and it was transfected into the mouse fibroblasts (NIH-3T3) cells. The expression of genes in pVITRO2-Mx-NA were measured by RT-PCR and indirect immunofluorescence assay (IFA). The recombinant plasmid was transfected into CEF cells then RT-PCR and the micro-cell inhibition tests were used to test the antiviral activity for NDV. Our results showed that co-expression vector pVITRO2-Mx-NA was constructed successfully; the expression of Mx and NA could be detected in both NIH-3T3 and CEF cells. The recombinant proteins of Mx and NA protect CEF cells from NDV infection until after 72 h of incubation but the individually mutagenic Mx protein or NA protein protects CEF cells from NDV infection till 48 h post-infection, and co-transfection group decreased significantly NDV infection compared with single-gene transfection group (P<0. 05), indicating that Mx-NA jointing contributed to delaying the infection of NDV in single-cell level and the co-transfection of the jointed genes was more powerful than single one due to their synergistic effects.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antibodies, Viral / immunology
  • Antiviral Agents / pharmacology*
  • Cell Shape / drug effects
  • Chickens / virology*
  • Fibroblasts / drug effects
  • Fibroblasts / pathology
  • Fibroblasts / virology
  • Fluorescent Antibody Technique
  • Gene Expression / drug effects
  • Mice
  • Myxovirus Resistance Proteins / genetics
  • Myxovirus Resistance Proteins / metabolism*
  • NIH 3T3 Cells
  • Neuraminidase / genetics*
  • Neuraminidase / metabolism
  • Newcastle disease virus / drug effects*
  • Newcastle disease virus / immunology
  • Plasmids / metabolism
  • RNA, Messenger / genetics
  • RNA, Messenger / metabolism
  • Recombinant Fusion Proteins / pharmacology*
  • Transfection

Substances

  • Antibodies, Viral
  • Antiviral Agents
  • Myxovirus Resistance Proteins
  • RNA, Messenger
  • Recombinant Fusion Proteins
  • Neuraminidase

Grants and funding

This work was supported by National Natural Science Foundation of China (31272429, 330871791); Research and Innovation Program for Graduate Cultivation of Jiangsu Proviance in 2010; Major Basic Research Program for Natural Science of Jiangsu Province Jiangsu Province; Specialized Research Grant for Doctoral Program (20103250110006); “Six personal peak” grant; “A Project Funded by the Priority Academic Program Development of Jiangsu Higher Education Institutions”. There are no any conflicts for above grant numbers. The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.