Tumor-associated edema in brain cancer patients: pathogenesis and management

Expert Rev Anticancer Ther. 2013 Nov;13(11):1319-25. doi: 10.1586/14737140.2013.852473. Epub 2013 Oct 23.

Abstract

The long-term treatment of peritumoral edema remains a major challenge in clinical neuro-oncology. Steroids have been and will remain the backbone of any anti-edematous therapy because of their striking activity, convenient oral administration and also because of their cost-effectiveness. Their side effects, however, can compromise quality of life, particularly upon continuous administration. Therapeutic alternatives which may replace or - at least - help to reduce the steroid dose are limited. However, with the development of new agents such as corticorelin acetate, there is a hope that steroid-induced side effects can be delayed and reduced. The administration of anti-angiogenic agents with steroid-sparing effects, for example, bevacizumab, is limited due to their costs. Increased knowledge on boswellic acids and cyclooxygenase-2 inhibitors which are available for clinical application may help to exploit their anti-edema activity more efficiently in the future.

Publication types

  • Review

MeSH terms

  • Brain Edema / drug therapy
  • Brain Edema / etiology*
  • Brain Edema / pathology
  • Brain Neoplasms / complications*
  • Brain Neoplasms / drug therapy
  • Brain Neoplasms / pathology
  • Cyclooxygenase 2 Inhibitors / adverse effects
  • Cyclooxygenase 2 Inhibitors / therapeutic use
  • Disease Management
  • Glucocorticoids / adverse effects
  • Glucocorticoids / therapeutic use
  • Humans

Substances

  • Cyclooxygenase 2 Inhibitors
  • Glucocorticoids