Abstract
Five patients with neuropathy and IgM M-proteins that reacted with myelin-associated glycoprotein (MAG) were treated for 10 to 20 months with cytostatic agents. In 2 patients, a decrease in serum M-protein and in anti-MAG IgM levels coincided with a progressive improvement of neuropathy. No clinical improvement and no decrease of anti-MAG IgM were observed in the other patients. The close relationship between the decrease of anti-MAG M-proteins and clinical improvement in these patients supports the pathogenetic role of the M-protein in the neuropathy.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Aged
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Antineoplastic Agents / therapeutic use*
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Autoantibodies / analysis*
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Blood Proteins / analysis*
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Chlorambucil / therapeutic use
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Cyclophosphamide / therapeutic use
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Humans
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Immunoglobulin M / analysis*
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Immunoglobulins*
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Male
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Middle Aged
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Myelin Proteins / immunology*
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Myelin-Associated Glycoprotein
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Neuromuscular Diseases / drug therapy*
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Neuromuscular Diseases / immunology*
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Peripheral Nervous System Diseases / drug therapy*
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Peripheral Nervous System Diseases / immunology
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Prednisone / therapeutic use
Substances
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Antineoplastic Agents
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Autoantibodies
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Blood Proteins
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Immunoglobulin M
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Immunoglobulins
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M-proteins (Myeloma)
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Myelin Proteins
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Myelin-Associated Glycoprotein
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Chlorambucil
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Cyclophosphamide
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Prednisone