Endothelin-1 participation in overt and inflammatory pain

J Cardiovasc Pharmacol. 1989:13 Suppl 5:S220-2. doi: 10.1097/00005344-198900135-00065.

Abstract

Endothelin-1 (ET-1) induced hyperalgesia in rats, abdominal constrictions in mice, incapacitation in dogs, and, when injected intradermally into humans, caused wheal and flare responses, which were accompanied by itching. The ET-1 induced constrictions in mice were prevented by indomethacin. In contrast, indomethacin had no effect on the hyperalgesic responses in the rat. These results indicate that ET-1 may play a role in modulating pain.

MeSH terms

  • Animals
  • Dogs
  • Dose-Response Relationship, Drug
  • Endothelins
  • Indomethacin / pharmacology
  • Inflammation / chemically induced*
  • Inflammation / complications
  • Injections, Intradermal
  • Mice
  • Pain / chemically induced*
  • Pain Measurement
  • Peptides / pharmacology*
  • Rats

Substances

  • Endothelins
  • Peptides
  • Indomethacin