Relative V beta transcript levels in thymus and peripheral lymphoid tissues from various mouse strains. Inverse correlation of I-E and Mls expression with relative abundance of several V beta transcripts in peripheral lymphoid tissues

J Exp Med. 1989 May 1;169(5):1703-19. doi: 10.1084/jem.169.5.1703.

Abstract

We have measured the relative levels of transcripts for 15 of the 22 known V beta gene segments. The level of transcripts for the highest and lowest expressed V beta gene segment differed by greater than 20-fold in the thymus and an even larger difference was observed in the periphery. The levels of expressions were unrelated to the order of the V beta genes on the chromosome. For most of the V beta gene segments, the relative transcript levels were the same in the thymus and periphery, suggesting that thymic selection in general does not act solely upon the V beta gene segment. One V beta gene segment in the BALB and B10 mice strains was an exception to this rule. V beta 5.2 expression in the periphery of BALB and B10 mice inversely correlated with the expression of the MHC class II molecule I-E. Five V beta gene segments had reduced transcript levels in the periphery of Mls-1a mice compared with their thymic levels or to the levels found in Mls-1b mice. The peripheral level of V beta 3 transcripts vary with MHC and Mls-2 haplotypes. The observation that certain V beta transcript levels are reduced in the periphery when compared with the thymus favors the hypothesis that self tolerance at the T cell level results in the elimination of self-reactive T cells, rather than paralysis by a block at some post-transcriptional step. Finally, the wide variability of V beta gene segment expression in the thymus suggests mechanisms exist to import an early bias to the repertoire. Whether this bias results from differential V beta segment rearrangement rates, differential V beta expression rates, or events occurring after TCR-alpha/beta expression on immature/nonmature thymocyte cell surfaces is yet to be determined.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Antigens, Surface / genetics*
  • Gene Expression Regulation*
  • Genes, Immunoglobulin
  • Haplotypes
  • Histocompatibility Antigens Class II / genetics*
  • Immunoglobulin Variable Region / genetics*
  • Lymphoid Tissue / immunology*
  • Major Histocompatibility Complex
  • Membrane Glycoproteins
  • Mice
  • Mice, Inbred AKR
  • Mice, Inbred BALB C
  • Mice, Inbred C57BL
  • Mice, Inbred CBA
  • Minor Lymphocyte Stimulatory Antigens
  • Receptors, Antigen, T-Cell / immunology
  • Ribonucleases
  • T-Lymphocytes / immunology
  • Thymus Gland / immunology*
  • Transcription, Genetic*

Substances

  • Antigens, Surface
  • Histocompatibility Antigens Class II
  • Immunoglobulin Variable Region
  • Membrane Glycoproteins
  • Minor Lymphocyte Stimulatory Antigens
  • Receptors, Antigen, T-Cell
  • Ribonucleases