Krebs cycle metabolite profiling for identification and stratification of pheochromocytomas/paragangliomas due to succinate dehydrogenase deficiency

J Clin Endocrinol Metab. 2014 Oct;99(10):3903-11. doi: 10.1210/jc.2014-2151. Epub 2014 Jul 11.

Abstract

Context: Mutations of succinate dehydrogenase A/B/C/D genes (SDHx) increase susceptibility to development of pheochromocytomas and paragangliomas (PPGLs), with particularly high rates of malignancy associated with SDHB mutations.

Objective: We assessed whether altered succinate dehydrogenase product-precursor relationships, manifested by differences in tumor ratios of succinate to fumarate or other metabolites, might aid in identifying and stratifying patients with SDHx mutations.

Design, setting, and patients: PPGL tumor specimens from 233 patients, including 45 with SDHx mutations, were provided from eight tertiary referral centers for mass spectrometric analyses of Krebs cycle metabolites.

Main outcome measure: Diagnostic performance of the succinate:fumarate ratio for identification of pathogenic SDHx mutations.

Results: SDH-deficient PPGLs were characterized by 25-fold higher succinate and 80% lower fumarate, cis-aconitate, and isocitrate tissue levels than PPGLs without SDHx mutations. Receiver-operating characteristic curves for use of ratios of succinate to fumarate or to cis-aconitate and isocitrate to identify SDHx mutations indicated areas under curves of 0.94 to 0.96; an optimal cut-off of 97.7 for the succinate:fumarate ratio provided a diagnostic sensitivity of 93% at a specificity of 97% to identify SDHX-mutated PPGLs. Succinate:fumarate ratios were higher in both SDHB-mutated and metastatic tumors than in those due to SDHD/C mutations or without metastases.

Conclusions: Mass spectrometric-based measurements of ratios of succinate:fumarate and other metabolites in PPGLs offer a useful method to identify patients for testing of SDHx mutations, with additional utility to quantitatively assess functionality of mutations and metabolic factors responsible for malignant risk.

Publication types

  • Multicenter Study
  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Adrenal Gland Neoplasms / enzymology
  • Adrenal Gland Neoplasms / genetics*
  • Adult
  • Aged
  • Citric Acid Cycle / genetics*
  • Citric Acid Cycle / physiology
  • Female
  • Follow-Up Studies
  • Fumarates / metabolism
  • Germ-Line Mutation
  • Humans
  • Male
  • Metabolic Diseases* / diagnosis
  • Metabolic Diseases* / enzymology
  • Metabolic Diseases* / genetics
  • Middle Aged
  • Paraganglioma / enzymology
  • Paraganglioma / genetics*
  • Pheochromocytoma / enzymology
  • Pheochromocytoma / genetics*
  • Sensitivity and Specificity
  • Succinate Dehydrogenase / deficiency
  • Succinate Dehydrogenase / genetics*
  • Succinate Dehydrogenase / metabolism
  • Succinic Acid / metabolism
  • Young Adult

Substances

  • Fumarates
  • Succinic Acid
  • Succinate Dehydrogenase