Abstract
We recently reported on the discovery of AMG 232, a potent and selective piperidinone inhibitor of the MDM2-p53 interaction. AMG 232 is being evaluated in human clinical trials for cancer. Continued exploration of the N-alkyl substituent of this series, in an effort to optimize interactions with the MDM2 glycine-58 shelf region, led to the discovery of sulfonamides such as compounds 31 and 38 that have similar potency, hepatocyte stability and rat pharmacokinetic properties to AMG 232.
Keywords:
MDM2; Piperidinone; Protein–protein interaction; Sulfonamide; p53.
Copyright © 2014 Elsevier Ltd. All rights reserved.
MeSH terms
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Acetates / chemistry
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Acetates / pharmacology*
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Animals
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Crystallography, X-Ray
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Dose-Response Relationship, Drug
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Drug Discovery*
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Drug Screening Assays, Antitumor
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Humans
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Models, Molecular
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Molecular Conformation
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Piperidones / chemistry
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Piperidones / pharmacology*
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Protein Binding / drug effects
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Proto-Oncogene Proteins c-mdm2 / antagonists & inhibitors*
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Proto-Oncogene Proteins c-mdm2 / chemistry
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Rats
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Structure-Activity Relationship
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Sulfonamides / chemistry*
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Tumor Suppressor Protein p53 / antagonists & inhibitors*
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Tumor Suppressor Protein p53 / chemistry
Substances
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2-(5-(3-chlorophenyl)-6-(4-chlorophenyl)-1-(1-(isopropylsulfonyl)-3-methylbutan-2-yl)-3-methyl-2-oxopiperidin-3-yl)acetic acid
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Acetates
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Piperidones
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Sulfonamides
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Tumor Suppressor Protein p53
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MDM2 protein, human
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Proto-Oncogene Proteins c-mdm2