Abstract
Compound {4-[({4-[(Z)-(2,4-dioxo-1,3-thiazolidin-5-ylidene)methyl]phenoxy}acetyl)amino]phenoxy}acetic acid (1) was prepared and the in vitro relative expression of PPARγ, GLUT-4 and PPARα, was estimated. Compound 1 showed an increase of 2-fold in the mRNA expression of PPARγ isoform, as well as the GLUT-4 levels. The antidiabetic activity of compound 1 was determined at 50 mg/Kg single dose using a non insulin dependent diabetes mellitus (NIDDM) rat model. The in vivo results indicated a significant decrease of plasma glucose levels, during the 7 h post-administration. Also, we performed a molecular docking of compound 1 into the ligand binding pocket of PPARγ, showing important short contacts with residues Ser289, His323 and His449 in the active site.
Keywords:
Diabetes; Molecular docking; PPAR.
Copyright © 2014 Elsevier Ltd. All rights reserved.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Animals
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Diabetes Mellitus, Experimental / drug therapy*
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Diabetes Mellitus, Experimental / metabolism
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Disease Models, Animal
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Glucose Transporter Type 4 / genetics
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Glucose Transporter Type 4 / metabolism*
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Humans
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Hypoglycemic Agents / chemical synthesis
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Hypoglycemic Agents / chemistry
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Hypoglycemic Agents / pharmacology*
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Hypoglycemic Agents / therapeutic use*
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Ligands
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Mice
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Molecular Docking Simulation
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Molecular Structure
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PPAR gamma / genetics
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PPAR gamma / metabolism*
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RNA, Messenger / genetics
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RNA, Messenger / metabolism
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Rats
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Thiazolidines / chemistry
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Thiazolidines / pharmacology*
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Thiazolidines / therapeutic use*
Substances
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4-(((4-((Z)-(2,4-dioxo-1,3-thiazolidin-5-ylidene)methyl)phenoxy)acetyl)amino)phenoxy)acetic acid
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Glucose Transporter Type 4
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Hypoglycemic Agents
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Ligands
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PPAR gamma
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RNA, Messenger
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Thiazolidines