Decreased lipases and fatty acid and glycerol transporter could explain reduced fat in diabetic morbidly obese

Obesity (Silver Spring). 2014 Nov;22(11):2379-87. doi: 10.1002/oby.20861. Epub 2014 Aug 6.

Abstract

Objective: The possible differences were investigated in 32 morbidly obese patients depending on whether they were "healthy" or had dyslipidemia and/or type 2 diabetes.

Methods: Lipid metabolism and insulin resistance were analyzed in subcutaneous (SAT) and visceral adipose tissue (VAT) before and during 6 and 12 months after Roux-en-Y gastric bypass.

Results: Significant differences have been found in lipoprotein lipase (LPL) and hormone-sensitive lipase (HSL) activities in SAT from the different obese group versus normal weight (control) but not between them. The reduced lipase activities in VAT were 43 and 19% smaller (22 and 4% smaller, respectively, vs. control) than the "healthy" obese group for LPL and HSL, respectively, and were accompanied with a reduced expression of these lipases, as well as decreased expression of FAT/CD36, FABP4, and AQ7 in that tissue. In addition, the expression of the other genes measured showed a downregulation not only versus the "healthy" obese but also versus the normal weight group.

Conclusions: Being obese is not "healthy," but it is even less so if morbidly obese patients with diabetes and dyslipidemia were considered. The reduced fat accumulation in these patients may be attributed to the decrease of the expression and activity of the lipases of their adipose tissue.

Publication types

  • Controlled Clinical Trial
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adiposity / genetics
  • Adult
  • Aquaporins / genetics
  • Aquaporins / metabolism
  • Biological Transport
  • CD36 Antigens* / genetics
  • CD36 Antigens* / metabolism
  • Diabetes Mellitus, Type 2 / complications
  • Diabetes Mellitus, Type 2 / genetics
  • Diabetes Mellitus, Type 2 / metabolism*
  • Diabetes Mellitus, Type 2 / surgery
  • Down-Regulation
  • Dyslipidemias / complications
  • Dyslipidemias / genetics
  • Dyslipidemias / metabolism
  • Dyslipidemias / pathology
  • Fatty Acid-Binding Proteins / genetics
  • Fatty Acid-Binding Proteins / metabolism
  • Fatty Acids / metabolism
  • Female
  • Glucose Transporter Type 4 / genetics
  • Glucose Transporter Type 4 / metabolism
  • Glycerol / metabolism*
  • Humans
  • Intra-Abdominal Fat* / metabolism
  • Intra-Abdominal Fat* / pathology
  • Lipase / genetics
  • Lipase / metabolism
  • Lipid Metabolism / genetics
  • Lipoprotein Lipase* / genetics
  • Lipoprotein Lipase* / metabolism
  • Male
  • Middle Aged
  • Obesity, Morbid / complications
  • Obesity, Morbid / genetics
  • Obesity, Morbid / metabolism*
  • Obesity, Morbid / surgery
  • Sterol Esterase* / genetics
  • Sterol Esterase* / metabolism
  • Subcutaneous Fat / metabolism
  • Subcutaneous Fat / pathology
  • Young Adult

Substances

  • AQP7 protein, human
  • Aquaporins
  • CD36 Antigens
  • FABP4 protein, human
  • Fatty Acid-Binding Proteins
  • Fatty Acids
  • Glucose Transporter Type 4
  • SLC2A4 protein, human
  • Sterol Esterase
  • Lipase
  • PNPLA2 protein, human
  • LPL protein, human
  • Lipoprotein Lipase
  • Glycerol