Nascent RNA interaction keeps PRC2 activity poised and in check

Genes Dev. 2014 Sep 15;28(18):1983-8. doi: 10.1101/gad.247940.114. Epub 2014 Aug 28.

Abstract

Polycomb-repressive complex 2 (PRC2) facilitates the maintenance and inheritance of chromatin domains repressive to transcription through catalysis of methylation of histone H3 at Lys27 (H3K27me2/3). However, through its EZH2 subunit, PRC2 also binds to nascent transcripts from active genes that are devoid of H3K27me2/3 in embryonic stem cells. Here, biochemical analyses indicated that RNA interaction inhibits SET domain-containing proteins, such as PRC2, nonspecifically in vitro. However, CRISPR-mediated truncation of a PRC2-interacting nascent RNA rescued PRC2-mediated deposition of H3K27me2/3. That PRC2 activity is inhibited by interactions with nascent transcripts supports a model in which PRC2 can only mark for repression those genes silenced by transcriptional repressors.

Keywords: EZH2; JARID2; PRC2; chromatin; ncRNA.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Clustered Regularly Interspaced Short Palindromic Repeats / physiology
  • Embryonic Stem Cells / cytology
  • Embryonic Stem Cells / metabolism
  • Enzyme Activation
  • Gene Expression Regulation
  • Gene Silencing
  • Histones / metabolism
  • Mice
  • Polycomb Repressive Complex 2 / genetics
  • Polycomb Repressive Complex 2 / metabolism*
  • Protein Binding
  • RNA / metabolism*
  • Sf9 Cells

Substances

  • Histones
  • Jarid2 protein, mouse
  • RNA
  • Polycomb Repressive Complex 2

Associated data

  • GEO/GSE60516