Abstract
Polymorphous low-grade adenocarcinoma (PLGA) is the second most frequent type of malignant tumor of the minor salivary glands. We identified PRKD1 hotspot mutations encoding p.Glu710Asp in 72.9% of PLGAs but not in other salivary gland tumors. Functional studies demonstrated that this kinase-activating alteration likely constitutes a driver of PLGA.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Adenocarcinoma / genetics*
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Adenocarcinoma / pathology
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Amino Acid Sequence
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Animals
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Humans
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Immunohistochemistry
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Immunoprecipitation
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Mice
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Microscopy, Confocal
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Models, Molecular*
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Molecular Sequence Data
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Mutagenesis
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Mutation, Missense / genetics
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NIH 3T3 Cells
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Protein Kinase C / chemistry
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Protein Kinase C / genetics*
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Salivary Gland Neoplasms / genetics*
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Salivary Gland Neoplasms / pathology
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Sequence Alignment
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Sequence Analysis, DNA
Substances
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protein kinase D
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Protein Kinase C
Associated data
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SRA/SRP045782
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SRA/SRP045785