Maternal nicotine exposure during gestation and lactation induces cardiac remodeling in rat offspring

Reprod Toxicol. 2014 Dec:50:4-10. doi: 10.1016/j.reprotox.2014.09.013. Epub 2014 Oct 2.

Abstract

The aim of this study was to evaluate the effects of maternal nicotine exposure on heart morphology and fibrosis in rat offspring. Nicotine was administered to pregnant Sprague-Dawley rats by using a subcutaneous osmotic mini-pump at a dose of 6 mg/kg/day from Gestational Days 7-21 or Gestational Day 7 to Postnatal Day 14. A control group received an equal volume of saline by the same route as nicotine. Rats born to prenatal nicotine-treated dams exhibited significantly greater cell width of cardiomyocytes, fewer cardiomyocyte nuclei number, higher β-myosin heavy chain and transforming growth factor (TGF-β1) expression, and higher collagen deposition in heart compared with rats born to normal saline-treated dams on Postnatal Days 7 and 21. Postnatal nicotine exposure further enhances these effects. We conclude that TGF-β1 may be involved in the pathogenesis of cardiac remodeling induced by maternal nicotine exposure during gestation and lactation in rat offspring.

Keywords: Collagen; Myosin heavy chain; Nicotine; Transforming growth factor-β1.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Body Weight / drug effects
  • Collagen / metabolism
  • Female
  • Fetal Heart / drug effects*
  • Fetal Heart / pathology
  • Lactation
  • Maternal Exposure*
  • Myosin Heavy Chains / analysis
  • Nicotine / toxicity*
  • Organ Size / drug effects
  • Pregnancy
  • Rats
  • Rats, Sprague-Dawley
  • Transforming Growth Factor beta1 / analysis

Substances

  • Transforming Growth Factor beta1
  • Nicotine
  • Collagen
  • Myosin Heavy Chains