Successful integration of nonclinical and clinical findings in interpreting the clinical relevance of rodent neoplasia with a new chemical entity

Toxicol Pathol. 2015 Jan;43(1):48-56. doi: 10.1177/0192623314557179. Epub 2014 Nov 14.

Abstract

Canagliflozin, a sodium glucose co-transporter 2 (SGLT2) inhibitor, has been developed for the treatment of adults with type 2 diabetes mellitus (T2DM). During the phase 3 program, treatment-related pheochromocytomas, renal tubular tumors, and testicular Leydig cell tumors were reported in the 2-year rat toxicology study. Treatment-related tumors were not seen in the 2-year mouse study. A cross-functional, mechanism-based approach was undertaken to determine whether the mechanisms responsible for tumorigenesis in the rat were of relevance to humans. Based on findings from nonclinical and clinical studies, the treatment-related tumors observed in rats were not deemed to be of clinical relevance. Here, we describe the scientific and regulatory journey from learning of the 2-year rat study findings to the approval of canagliflozin for the treatment of T2DM.

Keywords: Leydig cell tumors; SGLT2 inhibitor; canagliflozin; carbohydrate malabsorption.; pheochromocytomas; rat carcinogenicity; renal tubular tumors.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Canagliflozin / administration & dosage
  • Canagliflozin / toxicity*
  • Carcinogenicity Tests
  • Clinical Trials as Topic
  • Diabetes Mellitus, Type 2 / drug therapy
  • Humans
  • Hypoglycemic Agents / administration & dosage
  • Hypoglycemic Agents / toxicity*
  • Male
  • Mice
  • Neoplasms, Experimental / chemically induced*
  • Rats
  • Sodium-Glucose Transporter 2 Inhibitors
  • Testosterone

Substances

  • Hypoglycemic Agents
  • Sodium-Glucose Transporter 2 Inhibitors
  • Canagliflozin
  • Testosterone