Erythropoietin and resistant hypertension in CKD

Semin Nephrol. 2014;34(5):540-9. doi: 10.1016/j.semnephrol.2014.08.008.

Abstract

There is a well-documented association between erythropoiesis-stimulating agents (ESAs) and hypertension in chronic kidney disease. Studies suggest that the mechanism for this is multifactorial. First, some chronic kidney disease patients may have a limited ability to accommodate a rapid increase in red cell volume because of a decreased glomerular filtration rate, left ventricular hypertrophy, and decreased arterial compliance. Second, there is likely a direct vasoconstrictor effect of ESAs. Although no large randomized controlled trials of ESAs have been designed with blood pressure as an a priori outcome, several meta-analyses have explored this relationship and generally support the existence of ESA-induced hypertension. There are as of yet no data directly linking ESA-induced hypertension with increased cardiovascular morbidity and mortality. Despite this, clinicians should be vigilant for ESA-induced hypertension, use caution when using ESAs in patients with resistant hypertension, and be attentive to the rate of hemoglobin increase in patients with poorly controlled blood pressure.

Keywords: Erythropoietin-stimulating agents; anemia; chronic kidney disease; hypertension.

Publication types

  • Review

MeSH terms

  • Anemia / complications
  • Anemia / drug therapy*
  • Anemia / physiopathology
  • Blood Pressure / drug effects*
  • Drug Resistance
  • Endothelin-1 / metabolism
  • Erythropoietin / adverse effects*
  • Hematinics / adverse effects*
  • Humans
  • Hypertension / chemically induced*
  • Hypertension / physiopathology*
  • Nitric Oxide / metabolism
  • Recombinant Proteins / adverse effects
  • Renal Insufficiency, Chronic / complications*
  • Renin-Angiotensin System / physiology

Substances

  • Endothelin-1
  • Hematinics
  • Recombinant Proteins
  • Erythropoietin
  • Nitric Oxide