Endothelial glucocorticoid receptor suppresses atherogenesis--brief report

Arterioscler Thromb Vasc Biol. 2015 Apr;35(4):779-782. doi: 10.1161/ATVBAHA.114.304525. Epub 2015 Feb 19.

Abstract

Objective: The purpose of this study was to determine the role of the endothelial glucocorticoid receptor in the pathogenesis of atherosclerosis.

Approach and results: Control mice and mice lacking the endothelial glucocorticoid receptor were bred onto an Apoe knockout background and subjected to high-fat diet feeding for 12 weeks. Assessment of body weight and total cholesterol and triglycerides before and after the diet revealed no differences between the 2 groups of mice. However, mice lacking the endothelial glucocorticoid receptor developed more severe atherosclerotic lesions in the aorta, brachiocephalic artery, and aortic sinus, as well as a heightened inflammatory milieu as evidenced by increased macrophage recruitment in the lesions.

Conclusions: These data suggest that the endothelial glucocorticoid receptor is important for tonic inhibition of inflammation and limitation of atherosclerosis progression in this model.

Keywords: atherosclerosis; endothelium; glucocorticoid receptor.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Aorta / metabolism
  • Aorta / pathology
  • Aortic Diseases / blood
  • Aortic Diseases / genetics
  • Aortic Diseases / immunology
  • Aortic Diseases / pathology
  • Aortic Diseases / prevention & control*
  • Apolipoproteins E / deficiency
  • Apolipoproteins E / genetics
  • Atherosclerosis / blood
  • Atherosclerosis / genetics
  • Atherosclerosis / immunology
  • Atherosclerosis / pathology
  • Atherosclerosis / prevention & control*
  • Body Weight
  • Brachiocephalic Trunk / metabolism
  • Brachiocephalic Trunk / pathology
  • Cholesterol / blood
  • Diet, High-Fat
  • Disease Models, Animal
  • Endothelial Cells / metabolism*
  • Genotype
  • Macrophages / immunology
  • Mice, Inbred C57BL
  • Mice, Knockout
  • Phenotype
  • Receptors, Glucocorticoid / deficiency
  • Receptors, Glucocorticoid / genetics
  • Receptors, Glucocorticoid / metabolism*
  • Severity of Illness Index
  • Time Factors
  • Triglycerides / blood

Substances

  • Apolipoproteins E
  • NR3C1 protein, mouse
  • Receptors, Glucocorticoid
  • Triglycerides
  • Cholesterol