Studies on the relationship between protein kinase C and differentiation of human promyelocytic leukemia cells induced by retinoic acid

Leuk Res. 1989;13(10):869-74. doi: 10.1016/0145-2126(89)90039-8.

Abstract

We studied the differentiation of acute promyelocytic leukemia (APL) cells in 14 patients with APL. After the induction by retinoic acid (RA) the mature cells rose to 60 +/- 11.8% compared to 0.7 +/- 1% of the control, while the promyelocytes declined to 8.7 +/- 6.4% (93.3 +/- 5.6% in the control group). Protein kinase C (PKC) activity was significantly increased to 149.3 +/- 156.2 pmol/mg per min compared to 47 +/- 40.9 of the control (p less than 0.01). In HL-60 cells, the activity of PKC increased also from 52.3 +/- 35 to 129.2 +/- 64.6 pmol/mg per min (n = 10, p less than 0.01) after the induction of differentiation with RA. If the leukemia cells were pretreated with a kind of PKC inhibitor such as trifluoperazine, the increase of PKC activity was inhibited, and the rate of nitroblue tetrazolium reduction decreased from 89.9 +/- 7.7% to 62 +/- 25% (n = 6, p less than 0.01) and the mature cells reduced from 63.1 +/- 11.7% to 19.7 +/- 12.2% (p less than 0.01). We presumed that the activity of PKC is closely related to the differentiation of human promyelocytic leukemia cells induced by all-trans-retinoic acid.

MeSH terms

  • Adult
  • Cell Compartmentation / drug effects
  • Cell Differentiation / drug effects*
  • Female
  • Humans
  • Leukemia, Promyelocytic, Acute / enzymology
  • Leukemia, Promyelocytic, Acute / pathology*
  • Male
  • Protein Kinase C / physiology*
  • Tretinoin / pharmacology*
  • Trifluoperazine / pharmacology
  • Tumor Cells, Cultured

Substances

  • Trifluoperazine
  • Tretinoin
  • Protein Kinase C