miR-195a inhibits adipocyte differentiation by targeting the preadipogenic determinator Zfp423

J Cell Biochem. 2015 Nov;116(11):2589-97. doi: 10.1002/jcb.25204.

Abstract

MicroRNAs (miRNAs) play essential roles in various cellular processes including proliferation and differentiation. In this study, we identified miRNA-195a (miR-195a) as a regulator of adipocyte differentiation. Differential expression of miR-195a in preadipocytes and adipocytes suggests its role in lipid accumulation and adipocyte differentiation. Forced expression of miR-195a mimics suppressed lipid accumulation and inhibited expression of adipocyte markers such as PPARγ and aP2 in 3T3-L1 and C3H10T1/2 cells. Conversely, downregulation of miR-195a by anti-miR-195a increased lipid accumulation and expression of adipocyte markers. Target prediction analysis suggested zinc finger protein 423 (Zfp423), a preadipogenic determinator, as a potential gene recognized by miR-195a. In line with this, mimicked expression of miR-195a reduced the expression of Zfp423, whereas anti-miR-195a increased its expression. Predicted targeting sequences in Zfp423 3'UTR, but not mutated sequences fused to luciferase, were regulated by miR-195a. Ectopic Zfp423 expression in 3T3-L1 cells increased lipid accumulation and expression of adipocyte markers, consistent with the observation that miR-195a targets Zfp423, resulting in suppressed adipocyte differentiation. In addition, miR-195a and Zfp423 were inversely correlated in obese fat tissues, raising the possibility of miRNA's role in obesity. Together, our data show that miR-195a is an anti-adipogenic regulator, which acts by targeting Zfp423, and further suggest the roles of miR-195a in obesity and metabolic diseases.

Keywords: ADIPOCYTE DIFFERENTIATION; OBESITY; Zfp423; miR-195a.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • 3' Untranslated Regions
  • 3T3-L1 Cells
  • Adipocytes / cytology*
  • Adipocytes / metabolism
  • Animals
  • Cell Differentiation
  • DNA-Binding Proteins / genetics*
  • DNA-Binding Proteins / metabolism
  • Diet, High-Fat / adverse effects
  • Gene Expression Regulation
  • Lipid Metabolism
  • Mice
  • MicroRNAs / genetics*
  • MicroRNAs / metabolism*
  • Obesity / etiology
  • Obesity / genetics
  • Obesity / metabolism*
  • Promoter Regions, Genetic
  • Transcription Factors / genetics*
  • Transcription Factors / metabolism

Substances

  • 3' Untranslated Regions
  • DNA-Binding Proteins
  • Ebfaz protein, mouse
  • MIRN195a microRNA, mouse
  • MicroRNAs
  • Transcription Factors