Cox17 Protein Is an Auxiliary Factor Involved in the Control of the Mitochondrial Contact Site and Cristae Organizing System

J Biol Chem. 2015 Jun 12;290(24):15304-12. doi: 10.1074/jbc.M115.645069. Epub 2015 Apr 27.

Abstract

The mitochondrial contact site and cristae organizing system (MICOS) is a recently discovered protein complex that is crucial for establishing and maintaining the proper inner membrane architecture and contacts with the outer membrane of mitochondria. The ways in which the MICOS complex is assembled and its integrity is regulated remain elusive. Here, we report a direct link between Cox17, a protein involved in the assembly of cytochrome c oxidase, and the MICOS complex. Cox17 interacts with Mic60, thereby modulating MICOS complex integrity. This interaction does not involve Sco1, a partner of Cox17 in transferring copper ions to cytochrome c oxidase. However, the Cox17-MICOS interaction is regulated by copper ions. We propose that Cox17 is a newly identified factor involved in maintaining the architecture of the MICOS complex.

Keywords: Cox17; MICOS; Sco1; inner membrane architecture; membrane; metal; mitochondria; organelle; protein complex.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cation Transport Proteins / metabolism
  • Cation Transport Proteins / physiology*
  • Chromatography, Affinity
  • Copper Transport Proteins
  • Molecular Chaperones / metabolism
  • Molecular Chaperones / physiology*
  • Protein Binding
  • Saccharomyces cerevisiae / metabolism*
  • Saccharomyces cerevisiae Proteins / metabolism
  • Saccharomyces cerevisiae Proteins / physiology*

Substances

  • COX17 protein, S cerevisiae
  • Cation Transport Proteins
  • Copper Transport Proteins
  • Molecular Chaperones
  • Saccharomyces cerevisiae Proteins