Phase I trial of weekly MK-0752 in children with refractory central nervous system malignancies: a pediatric brain tumor consortium study

Childs Nerv Syst. 2015 Aug;31(8):1283-9. doi: 10.1007/s00381-015-2725-3. Epub 2015 May 1.

Abstract

Purpose: Amplification and high levels of NOTCH ligand expression have been identified in several types of pediatric brain tumors. A phase I trial of weekly MK-0752, an oral inhibitor of gamma-secretase, was conducted in children with recurrent central nervous system (CNS) malignancies to estimate the maximum tolerated dose, dose-limiting toxicities (DLT), pharmacokinetics (PK), and pharmacodynamics of weekly MK-0752.

Methods: MK-0752 was administered once weekly at 1000 and 1400 mg/m(2) using a rolling-6 design. PK analysis was performed during the first course. NOTCH and HES expression was assessed by immunohistochemistry and Western blot.

Results: Ten eligible patients were enrolled (median age 8.8 years; range 3.1-19.2) with diagnoses of brain stem glioma (n = 3), ependymoma (n = 2), anaplastic astrocytoma (n = 1), choroid plexus carcinoma (n = 2), medulloblastoma (n = 1), and primitive neuroectodermal tumor (n = 1). Nine were evaluable for toxicity. One DLT of fatigue occurred in the six evaluable patients enrolled at 1000 mg/m(2)/dose. No DLTs were experienced by three patients treated at 1400 mg/m(2)/dose. Non-dose-limiting grade 3 toxicities included lymphopenia, neutropenia, and anemia. Median number of treatment courses was 2 (range 1-10). Two patients continued on therapy for at least 6 months. The median (range) C(max) of MK-0752 was 88.2 μg/mL (40.6 to 109 μg/mL) and 60.3 μg/mL (59.2 to 91.9 μg/mL) in patients receiving 1000 and 1400 mg/m(2)/week, respectively. NOTCH expression was decreased in six of seven patients for whom tissue was available at 24 h post-MK-0752.

Conclusion: MK-0752 is well tolerated and exhibits target inhibition at 1000 and 1400 mg/m(2)/week in children with recurrent CNS malignancies.

Publication types

  • Clinical Trial, Phase I
  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Administration, Oral
  • Adolescent
  • Amyloid Precursor Protein Secretases / antagonists & inhibitors
  • Area Under Curve
  • Basic Helix-Loop-Helix Transcription Factors / metabolism
  • Benzene Derivatives / blood
  • Benzene Derivatives / therapeutic use*
  • Brain Neoplasms / drug therapy*
  • Central Nervous System Diseases / blood
  • Central Nervous System Diseases / drug therapy*
  • Child
  • Child, Preschool
  • Dose-Response Relationship, Drug
  • Enzyme Inhibitors / blood
  • Enzyme Inhibitors / therapeutic use*
  • Female
  • Follow-Up Studies
  • Gene Expression Regulation / drug effects
  • Homeodomain Proteins / metabolism
  • Humans
  • Male
  • Propionates / blood
  • Propionates / therapeutic use*
  • Receptor, Notch1 / metabolism
  • Repressor Proteins / metabolism
  • Sulfones / blood
  • Sulfones / therapeutic use*
  • Time Factors
  • Transcription Factor HES-1
  • Young Adult

Substances

  • 3-(4-((4-chlorophenyl)sulfonyl)-4-(2,5-difluorophenyl)cyclohexyl)propanoic acid
  • Basic Helix-Loop-Helix Transcription Factors
  • Benzene Derivatives
  • Enzyme Inhibitors
  • Homeodomain Proteins
  • NOTCH1 protein, human
  • Propionates
  • Receptor, Notch1
  • Repressor Proteins
  • Sulfones
  • Transcription Factor HES-1
  • HES5 protein, human
  • HES1 protein, human
  • Amyloid Precursor Protein Secretases