β3-Adrenoceptor, glutathionylation, and diabetic cardiomyopathy. Focus on "β3-Adrenoceptor activation relieves oxidative inhibition of the cardiac Na+-K+ pump in hyperglycemia induced by insulin receptor blockade"

Am J Physiol Cell Physiol. 2015 Sep 1;309(5):C283-5. doi: 10.1152/ajpcell.00196.2015. Epub 2015 Jul 15.
No abstract available

Publication types

  • Editorial
  • Research Support, N.I.H., Extramural

MeSH terms

  • Adrenergic beta-Agonists / administration & dosage
  • Animals
  • Diabetic Cardiomyopathies / drug therapy
  • Diabetic Cardiomyopathies / metabolism*
  • Drug Delivery Systems / trends
  • Glutathione / metabolism
  • Humans
  • Hyperglycemia / drug therapy
  • Hyperglycemia / metabolism*
  • Oxidative Stress / drug effects
  • Oxidative Stress / physiology*
  • Receptor, Insulin / antagonists & inhibitors*
  • Receptors, Adrenergic, beta-3 / metabolism*
  • Sodium-Potassium-Exchanging ATPase / antagonists & inhibitors*

Substances

  • Adrenergic beta-Agonists
  • Receptors, Adrenergic, beta-3
  • Receptor, Insulin
  • Sodium-Potassium-Exchanging ATPase
  • Glutathione