miR-107 promotes hepatocellular carcinoma cell proliferation by targeting Axin2

Int J Clin Exp Pathol. 2015 May 1;8(5):5168-74. eCollection 2015.

Abstract

Background: A large number of studies demonstrated that microRNAs play important roles in the progression and development of human cancers. However, the expression level of miR-107 and its biological function in hepatocellular carcinoma (HCC) remains unclear.

Method: Quantitative real-time PCR (qRT-PCR) was used to evaluate the expression level of miR-107 in HCC tissues and cell lines. Then, we explored the function of miR-107 to determine its potential roles on HCC cell proliferation in vitro. Luciferase reporter assay was used to confirm the target gene of miR-107, and the results were validated in cell lines.

Results: miR-107 was significantly up-regulated in HCC tissues and cell lines. The enforced expression of miR-107 was able to promote cell proliferation in HepG2 cells. At the molecular level, our results suggested that expression of Axin2 was negatively regulated by miR-107.

Conclusion: Our observations suggested that miR-107 could promote HCC cells proliferation via targeting Axin2 and might represent a potential therapeutic target for HCC.

Keywords: Axin2; Hepatocellular carcinoma; miR-107; proliferation.

MeSH terms

  • Axin Protein / genetics
  • Axin Protein / metabolism*
  • Carcinoma, Hepatocellular / genetics
  • Carcinoma, Hepatocellular / metabolism*
  • Carcinoma, Hepatocellular / pathology
  • Cell Proliferation*
  • Gene Expression Regulation, Neoplastic
  • Genes, Reporter
  • Hep G2 Cells
  • Humans
  • Liver Neoplasms / genetics
  • Liver Neoplasms / metabolism*
  • Liver Neoplasms / pathology
  • Luciferases / genetics
  • Luciferases / metabolism
  • MicroRNAs / genetics
  • MicroRNAs / metabolism*
  • Signal Transduction
  • Time Factors
  • Transfection

Substances

  • AXIN2 protein, human
  • Axin Protein
  • MIRN107 microRNA, human
  • MicroRNAs
  • Luciferases