Two brothers homozygous for the TTR V30M both presenting with a phenotype dominated by central nervous complications
Amyloid
.
2015;22(4):261-2.
doi: 10.3109/13506129.2015.1104661.
Epub 2015 Nov 20.
Authors
Yuto Uchida
1
,
Koji Takada
1
,
Yasukuni Tsugu
1
,
Mitsuharu Ueda
2
,
Taro Yamashita
2
,
Yukio Ando
2
,
Susumu Kobayashi
3
,
Haruki Koike
4
,
Takayuki Watanabe
5
,
Takashi Matsumoto
5
,
Takanari Toyoda
6
,
Gohei Yamada
6
,
Noriyuki Matsukawa
6
Affiliations
1
a Department of Neurology , Toyokawa City Hospital , 23 Noji, Yawata-cho, Toyokawa 442-8561, Aichi , Japan .
2
b Department of Neurology , Kumamoto University , Kumamoto , Japan .
3
c Department of Radiology , Toyokawa City Hospital , Aichi , Japan .
4
d Department of Neurology , Nagoya University , Aichi , Japan .
5
e Department of Neurosurgery , Toyokawa City Hospital , Aichi , Japan , and.
6
f Department of Neurology , Nagoya City University , Aichi , Japan.
PMID:
26587769
DOI:
10.3109/13506129.2015.1104661
No abstract available
Publication types
Case Reports
Letter
MeSH terms
Cerebral Amyloid Angiopathy / blood
Cerebral Amyloid Angiopathy / genetics*
Homozygote*
Humans
Magnetic Resonance Imaging
Male
Mass Spectrometry
Mutation*
Phenotype
Prealbumin / genetics*
Siblings*
Substances
Prealbumin
Supplementary concepts
Amyloid angiopathy