Acute Respiratory Distress Syndrome: Role of Oleic Acid-Triggered Lung Injury and Inflammation

Mediators Inflamm. 2015:2015:260465. doi: 10.1155/2015/260465. Epub 2015 Nov 12.

Abstract

Lung injury especially acute respiratory distress syndrome (ARDS) can be triggered by diverse stimuli, including fatty acids and microbes. ARDS affects thousands of people worldwide each year, presenting high mortality rate and having an economic impact. One of the hallmarks of lung injury is edema formation with alveoli flooding. Animal models are used to study lung injury. Oleic acid-induced lung injury is a widely used model resembling the human disease. The oleic acid has been linked to metabolic and inflammatory diseases; here we focus on lung injury. Firstly, we briefly discuss ARDS and secondly we address the mechanisms by which oleic acid triggers lung injury and inflammation.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Animals
  • Cystic Fibrosis Transmembrane Conductance Regulator / physiology
  • Disease Models, Animal
  • Humans
  • Inflammation / chemically induced*
  • Inflammation / complications
  • Inflammation Mediators / physiology
  • Lung Injury / chemically induced*
  • Lung Injury / complications
  • Oleic Acid / toxicity*
  • Pulmonary Edema / physiopathology
  • Respiratory Distress Syndrome / etiology*
  • Sodium-Potassium-Exchanging ATPase / antagonists & inhibitors

Substances

  • Inflammation Mediators
  • Cystic Fibrosis Transmembrane Conductance Regulator
  • Oleic Acid
  • Sodium-Potassium-Exchanging ATPase