Thrombopoietin/TGF-β1 Loop Regulates Megakaryocyte Extracellular Matrix Component Synthesis

Stem Cells. 2016 Apr;34(4):1123-33. doi: 10.1002/stem.2285. Epub 2016 Jan 29.

Abstract

Extracellular matrix (ECM) components initiate crucial biochemical and biomechanical cues that are required for bone marrow homeostasis. In our research, we prove that a peri-cellular matrix composed primarily of type III and type IV collagens, and fibronectin surrounds human megakaryocytes in the bone marrow. The data we collected support the hypothesis that bone marrow megakaryocytes possess a complete mechanism to synthesize the ECM components, and that thrombopoietin is a pivotal regulator of this new function inducing transforming growth factor-β1 (TGF-β1) release and consequent activation of the downstream pathways, both in vitro and in vivo. This activation results in a dose dependent increase of ECM component synthesis by megakaryocytes, which is reverted upon incubation with JAK and TGF-β1 receptor specific inhibitors. These data are pivotal for understanding the central role of megakaryocytes in creating their own regulatory niche within the bone marrow environment.

Keywords: Bone marrow; Extracellular matrix; Megakaryocytes; Thrombopoietin; Transforming growth factor-β1.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Bone Marrow / growth & development
  • Bone Marrow / metabolism
  • Collagen Type III / metabolism
  • Collagen Type IV / metabolism
  • Extracellular Matrix / genetics
  • Extracellular Matrix / metabolism*
  • Fetal Blood / cytology
  • Fetal Blood / metabolism
  • Fibronectins / metabolism
  • Humans
  • Janus Kinases / antagonists & inhibitors
  • Janus Kinases / metabolism
  • Megakaryocytes / drug effects
  • Megakaryocytes / metabolism*
  • Mice
  • Receptors, Transforming Growth Factor beta / antagonists & inhibitors
  • Receptors, Transforming Growth Factor beta / metabolism
  • Thrombopoietin / genetics
  • Thrombopoietin / metabolism*
  • Transforming Growth Factor beta1 / genetics
  • Transforming Growth Factor beta1 / metabolism*

Substances

  • Collagen Type III
  • Collagen Type IV
  • Fibronectins
  • Receptors, Transforming Growth Factor beta
  • Transforming Growth Factor beta1
  • Thrombopoietin
  • Janus Kinases