Conformation of NAD+ bound to allosteric L-lactate dehydrogenase activated by chemical modification

J Biol Chem. 1989 May 25;264(15):8676-9.

Abstract

On modification of arginine residues with 2,3-butanedione, the Thermus caldophilus L-lactate dehydrogenase is converted to an activated form that is independent of an allosteric effector, fructose 1,6-bisphosphate (Fru-1,6-P2). The conformation of NAD+ bound to the modified enzyme in the absence of Fru-1,6-P2 was investigated by means of proton NMR, analyzing the time dependence of the transferred nuclear Overhauser effect (TRNOE) and TRNOE action spectra. The inter-proton distances determined on TRNOE analysis indicated that both the nicotinamide riboside moiety and the adenosine moiety of NAD+ were in the anti conformation, the ribose rings being in the C3'-endo form. This conformation was almost the same as that of NAD+ bound to the native enzyme-Fru-1,6-P2 complex, rather than that of NAD+ bound to the free native enzyme. These results suggest that the C3'-endo-anti form of the enzyme-bound NAD+ is essential for the activation of the T. caldophilus L-lactate dehydrogenase.

MeSH terms

  • Allosteric Regulation
  • Allosteric Site
  • Arginine
  • Butanones / pharmacology*
  • Diacetyl / pharmacology*
  • Fructosediphosphates / pharmacology*
  • Hexosediphosphates / pharmacology*
  • Kinetics
  • L-Lactate Dehydrogenase / metabolism*
  • Magnetic Resonance Spectroscopy
  • Models, Molecular
  • Molecular Conformation
  • NAD / metabolism*
  • Thermus / enzymology

Substances

  • Butanones
  • Fructosediphosphates
  • Hexosediphosphates
  • NAD
  • Arginine
  • L-Lactate Dehydrogenase
  • Diacetyl
  • fructose-1,6-diphosphate