Can Bias Evaluation Provide Protection Against False-Negative Results in QT Studies Without a Positive Control Using Exposure-Response Analysis?

J Clin Pharmacol. 2017 Jan;57(1):85-95. doi: 10.1002/jcph.779. Epub 2016 Jul 7.

Abstract

The revised ICH E14 document allows the use of exposure-response analysis to exclude a small QT effect of a drug. If plasma concentrations exceeding clinically relevant levels is achieved, a positive control is not required. In cases when this cannot be achieved, there may be a need for metrics to protect against false-negative results. The objectives of this study were to create bias in electrocardiogram laboratory QT-interval measurements and define a metric that can be used to detect bias severe enough to cause false-negative results using exposure-response analysis. Data from the IQ-CSRC study, which evaluated the QT effect of 5 QT-prolonging drugs, were used. Negative bias using 3 deterministic and 2 random methods was introduced into the reported QTc values and compared with fully automated data from the underlying electrocardiogram algorithm (COMPAS). The slope estimate of the Bland-Altman plot was used as a bias metric. With the deterministic bias methods, negative bias, measured between electrocardiogram laboratory values and COMPAS, had to be larger than approximately -20 milliseconds over a QTcF range of 100 milliseconds to cause failures to predict the QT effect of ondansetron, quinine, dolasetron, moxifloxacin, and dofetilide. With the random methods, the rate of false-negatives was ≤5% with bias severity < -10 milliseconds for all 5 drugs when plasma levels exceeded those of interest. Severe and therefore detectable bias has to be introduced into reported QTc values to cause false-negative predictions with exposure-response analysis.

Keywords: QT; bias; early phase; exposure response analysis; first-in-human; positive control.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cardiotoxins / administration & dosage
  • Cardiotoxins / adverse effects*
  • Dose-Response Relationship, Drug
  • Electrocardiography / drug effects*
  • Electrocardiography / methods
  • Heart Conduction System / drug effects
  • Heart Conduction System / physiopathology
  • Heart Rate / drug effects
  • Heart Rate / physiology
  • Humans
  • Long QT Syndrome / chemically induced*
  • Long QT Syndrome / diagnosis*
  • Long QT Syndrome / physiopathology

Substances

  • Cardiotoxins