Systemic therapy in metastatic renal cell carcinoma

World J Urol. 2017 Feb;35(2):179-188. doi: 10.1007/s00345-016-1868-5. Epub 2016 Jun 9.

Abstract

Purpose: Current systemic treatment of targeted therapies, namely the vascular endothelial growth factor-antibody (VEGF-AB), VEGF receptor tyrosine kinase inhibitor (TKI) and mammalian target of rapamycin (mTOR) inhibitors, have improved progression-free survival and replaced non-specific immunotherapy with cytokines in metastatic renal cell carcinoma (mRCC).

Methods: A panel of experts convened to review currently available phase 3 data for mRCC treatment of approved agents, in addition to available EAU guideline data for a collaborative review as the plurality of substances offers different options of first-, second- and third-line treatment with potential sequencing.

Results: Sunitinib and pazopanib are approved treatments in first-line therapy for patients with favorable- or intermediate-risk clear cell RCC (ccRCC). Temsirolimus has proven benefit over interferon-alfa (IFN-α) in patients with non-clear cell RCC (non-ccRCC). In the second-line treatment TKIs or mTOR inhibitors are treatment choices. Therapy options after TKI failure consist of everolimus and axitinib. Available third-line options consist of everolimus and sorafenib. Recently, nivolumab, a programmed death-1 (PD1) checkpoint inhibitor, improved overall survival benefit compared to everolimus after failure of one or two VEGFR-targeted therapies, which is likely to become the first established checkpoint inhibitor in mRCC. Data for the sequencing of agents remain limited.

Conclusions: Despite the high level of evidence for first and second-line treatment in mRCC, data for third-line therapy are limited. Possible sequences include TKI-mTOR-TKI or TKI-TKI-mTOR with the upcoming checkpoint inhibitors in perspective, which might settle a new standard of care after previous TKI therapy.

Keywords: Checkpoint inhibitor; Renal cell carcinoma; Sequence; Systemic treatment; Targeted therapy; Tyrosine kinase inhibitor mTOR inhibition.

Publication types

  • Review

MeSH terms

  • Antibodies, Monoclonal / therapeutic use
  • Antineoplastic Agents / therapeutic use*
  • Axitinib
  • Carcinoma, Renal Cell / drug therapy*
  • Decision Trees
  • Everolimus / therapeutic use
  • Humans
  • Imidazoles / therapeutic use
  • Indazoles / therapeutic use
  • Indoles / therapeutic use
  • Niacinamide / analogs & derivatives
  • Niacinamide / therapeutic use
  • Nivolumab
  • Phenylurea Compounds / therapeutic use
  • Pyrimidines / therapeutic use
  • Pyrroles / therapeutic use
  • Sirolimus / analogs & derivatives
  • Sirolimus / therapeutic use
  • Sorafenib
  • Sulfonamides / therapeutic use
  • Sunitinib

Substances

  • Antibodies, Monoclonal
  • Antineoplastic Agents
  • Imidazoles
  • Indazoles
  • Indoles
  • Phenylurea Compounds
  • Pyrimidines
  • Pyrroles
  • Sulfonamides
  • Niacinamide
  • Nivolumab
  • temsirolimus
  • pazopanib
  • Everolimus
  • Sorafenib
  • Axitinib
  • Sunitinib
  • Sirolimus

Supplementary concepts

  • Clear-cell metastatic renal cell carcinoma