Macular Ganglion Cell -Inner Plexiform Layer Thickness Is Associated with Clinical Progression in Mild Cognitive Impairment and Alzheimers Disease

PLoS One. 2016 Sep 6;11(9):e0162202. doi: 10.1371/journal.pone.0162202. eCollection 2016.

Abstract

Purpose: We investigated the association of the macular ganglion cell-inner plexiform layer (GCIPL) and peripapillary retinal nerve fiber layer (RNFL) thicknesses with disease progression in mild cognitive impairment (MCI) and Alzheimer's disease (AD).

Methods: We recruited 42 patients with AD, 26 with MCI, and 66 normal elderly controls. The thicknesses of the RNFL and GCIPL were measured via spectral-domain optic coherent tomography in all participants at baseline. The patients with MCI or AD underwent clinical and neuropsychological tests at baseline and once every year thereafter for 2 years.

Results: The Clinical Dementia Rating scale-Sum of Boxes (CDR-SB) score exhibited significant negative relationships with the average GCIPL thickness (β = -0.15, p < 0.05) and the GCIPL thickness in the superotemporal, superonasal, and inferonasal sectors. The composite memory score exhibited significant positive associations with the average GCIPL thickness and the GCIPL thickness in the superotemporal, inferonasal, and inferotemporal sectors. The temporal RNFL thickness, the average and minimum GCIPL thicknesses, and the GCIPL thickness in the inferonasal, inferior, and inferotemporal sectors at baseline were significantly reduced in MCI patients who were converted to AD compared to stable MCI patients. The change of CDR-SB from baseline to 2 years exhibited significant negative associations with the average (β = -0.150, p = 0.006) and minimum GCIPL thicknesses as well as GCIPL thickness in the superotemporal, superior, superonasal, and inferonasal sectors at baseline.

Conclusions: Our data suggest that macular GCIPL thickness represents a promising biomarker for monitoring the progression of MCI and AD.

MeSH terms

  • Aged
  • Aged, 80 and over
  • Alzheimer Disease / diagnostic imaging*
  • Alzheimer Disease / pathology
  • Alzheimer Disease / physiopathology
  • Biomarkers / analysis
  • Case-Control Studies
  • Cognitive Dysfunction / diagnostic imaging*
  • Cognitive Dysfunction / pathology
  • Cognitive Dysfunction / physiopathology
  • Disease Progression
  • Female
  • Humans
  • Male
  • Memory / physiology
  • Middle Aged
  • Nerve Fibers / pathology*
  • Optic Nerve / diagnostic imaging*
  • Optic Nerve / pathology
  • Optic Nerve / physiopathology
  • Retinal Ganglion Cells / pathology*
  • Tomography, Optical Coherence

Substances

  • Biomarkers

Grants and funding

This work was supported by the Basic Science Research Program through the National Research Foundation of Korea (NRF) funded by the Ministry of Science, ICT, and Future Planning (No. 2016R1A1A1A05005484) and by the Original Technology Research Program for Brain Science through the NRF funded by the Korean government (MSIP) (No. 2014M3C7A1064752). The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.