Pregnancy-driven cardiovascular maternal miR-29 plasticity in obesity

J Med Primatol. 2016 Dec;45(6):297-303. doi: 10.1111/jmp.12236. Epub 2016 Sep 15.

Abstract

Background: Obesity in pregnancy (MO) is a risk factor for maternal and/or fetal cardiovascular system disorders. This study evaluated maternal CVS expression of microRNA-29 family and its target molecules in MO to test the hypotheses: CVS miR-29 concentrations are increased in pregnancy and decreased in MO.

Methods: Non-pregnant (n=4), pregnant obese (POb, n=4), and pregnant non-obese (PnOb, n=4) baboons (Papio spp.) were studied. Maternal left ventricle (LV), left atrium (LA), and aortic arch (AA) were collected at the end of gestation. Expression of MiR-29 and elastin (ELN) mRNA were quantified.

Results: LA miR-29 (a, c) expression was highest in PnOb. In the LV, miR-29b expression trended lower (P=.059) for PnOb animals. ELN mRNA expression correlated positively with miR-29b expression in AA (r=.76, P=.03).

Conclusion: Maternal obesity diminishes miR-29 adaptation to pregnancy. Pharmacologic, tissue-specific targeting of miRNA-29 may represent a strategy for prevention and treatment of MO complications.

Keywords: maternal obesity; non-human primate; vascular remodeling.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Animals
  • Female
  • MicroRNAs / metabolism*
  • Monkey Diseases / etiology
  • Monkey Diseases / metabolism*
  • Myocardium / metabolism
  • Obesity / etiology
  • Obesity / metabolism
  • Obesity / veterinary*
  • Papio*
  • Pregnancy

Substances

  • MicroRNAs