The Effect of Prophylactic Lamivudine plus Adefovir Therapy Compared with Lamivudine Alone in Preventing Hepatitis B Reactivation in Lymphoma Patients with High Baseline HBV DNA during Chemotherapy

PLoS One. 2016 Oct 6;11(10):e0164210. doi: 10.1371/journal.pone.0164210. eCollection 2016.

Abstract

Prophylactic antiviral therapy is essential for lymphoma patients with high baseline HBV DNA who undergo cytotoxic chemotherapy. However, there are limited data on the optimal options. The present study was designed to compare the efficacy of prophylactic lamivudine (LAM) with lamivudine plus adefovir dipivoxil (LAM+ADV) in preventing hepatitis B virus (HBV) reactivation in lymphoma with, pre-chemotherapy HBV DNA load ≥2000 IU/ml. We retrospectively analyzed the medical records of 86 lymphoma patients with baseline HBV DNA load ≥2000 IU/ml during chemotherapy and received LAM or LAM+ADV as prophylaxis between January 1, 2008 and November 30, 2014 at Sun Yat-sen University Cancer Center, China. Sixty-five patients received LAM and 21 received LAM+ADV. The rate was significantly lower in the LAM+ADV group compared with the LAM group for HBV reactivation (23.8% vs 55.4%; p = 0.012), while no difference was observed between the two groups in patients for HBV-related hepatitis (21.3% vs 33.3%; p = 0.349), and chemotherapy disruption (10.9% vs 19.0%; p = 0.337). In a multivariate analysis of factors associated with HBV reactivation in these patients, LAM+ADV treatment and HBeAg negative were the independent protective factors. Therefore, LAM+ADV should be considered for antiviral prophylaxis in lymphoma patients with pre-chemotherapy HBV DNA load ≥2000 IU/ml. Further study is warranted to confirm these findings.

Publication types

  • Comparative Study

MeSH terms

  • Adenine / analogs & derivatives*
  • Adenine / pharmacology
  • Adolescent
  • Adult
  • Aged
  • Aged, 80 and over
  • Antiviral Agents / pharmacology
  • Child
  • DNA, Viral / metabolism*
  • Drug Interactions
  • Female
  • Hepatitis B virus / drug effects*
  • Hepatitis B virus / genetics
  • Hepatitis B virus / physiology
  • Humans
  • Lamivudine / pharmacology*
  • Lymphoma / drug therapy*
  • Lymphoma / virology*
  • Male
  • Middle Aged
  • Organophosphonates / pharmacology*
  • Retrospective Studies
  • Risk
  • Virus Activation / drug effects*
  • Young Adult

Substances

  • Antiviral Agents
  • DNA, Viral
  • Organophosphonates
  • Lamivudine
  • adefovir
  • Adenine

Grants and funding

The work was supported by National Natural Science Foundation of China (NO. 81672686, NO. 81372883, NO. 81001052), Natural Science Foundation of Guangdong Province, China (2015A030313020), Science and Technology Planning Project of Guangdong Province, China (2011B031800222), Young Talents Project of Sun Yat-sen University Cancer Center (to Qingqing Cai), Young Talents Key Project of Sun Yat-sen University (2015ykzd13, to Qingqing Cai), and Sister Institution Network Fund of MD Anderson Cancer Center (to Qingqing Cai; Huilan Rao).