Abstract
The in vitro activities of fungal CYP51 inhibitors VT-1161 and VT-1129 were determined for Candida glabrata (n = 34) and C. krusei (n = 50). C. glabrata isolates were screened for FKS gene mutations. All isolates were resistant clinically and/or in vitro to at least one standard antifungal compound. VT-1161 and VT-1129 MICs for all isolates were at least 5-fold below achievable human plasma levels for VT-1161. VT-1161 and VT-1129 are promising for the treatment of resistant C. glabrata and C. krusei infections.
Keywords:
Candida glabrata; Candida krusei; antifungal susceptibility testing; resistance.
Copyright © 2017 American Society for Microbiology.
MeSH terms
-
14-alpha Demethylase Inhibitors / pharmacology*
-
Antifungal Agents / pharmacology*
-
Azoles / pharmacology
-
Candida / drug effects*
-
Candida / genetics
-
Candida / growth & development
-
Candida / isolation & purification
-
Candida glabrata / drug effects
-
Candida glabrata / genetics
-
Candida glabrata / growth & development
-
Candida glabrata / isolation & purification
-
Candidiasis / drug therapy
-
Candidiasis / microbiology
-
Drug Resistance, Fungal / drug effects*
-
Echinocandins / pharmacology
-
Fungal Proteins / genetics
-
Fungal Proteins / metabolism
-
Gene Expression
-
Glucosyltransferases / genetics
-
Glucosyltransferases / metabolism
-
Humans
-
Microbial Sensitivity Tests
-
Mutation
-
Pyridines / pharmacology*
-
Tetrazoles / pharmacology*
Substances
-
14-alpha Demethylase Inhibitors
-
Antifungal Agents
-
Azoles
-
Echinocandins
-
Fungal Proteins
-
Pyridines
-
Tetrazoles
-
VT-1129
-
VT-1161
-
Glucosyltransferases