Corticosteroids and infliximab impair the performance of interferon-γ release assays used for diagnosis of latent tuberculosis

Thorax. 2017 Oct;72(10):946-949. doi: 10.1136/thoraxjnl-2016-209397. Epub 2017 Feb 3.

Abstract

The impact of immunosuppression on interferon-γ release assays and novel cytokine biomarkers of TB infection, mycobacteria-specific IL-2, IP-10 and TNF-α responses was investigated in an ex vivo model. Cytokine responses in standard QuantiFERON-TB Gold in-Tube (QFT-GIT) assays were compared with duplicate assays containing dexamethasone or infliximab. Dexamethasone converted QFT-GIT results from positive to negative in 30% of participants. Antigen-stimulated interferon-γ, IL-2 and TNF-α responses were markedly reduced, but IP-10 responses were preserved. Infliximab caused QFT-GIT result conversion in up to 30% of participants and substantial reductions in all cytokine responses. Therefore, corticosteroids and anti-TNF-α agents significantly impair interferon-γ release assay performance. IP-10 may be a more robust TB biomarker than interferon-γ in patients receiving corticosteroids.

Keywords: Tuberculosis.

Publication types

  • Letter
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adrenal Cortex Hormones / pharmacology*
  • Adult
  • Aged
  • Antirheumatic Agents / pharmacology*
  • Dexamethasone / pharmacology
  • Female
  • Humans
  • Infliximab / pharmacology*
  • Interferon-gamma / metabolism
  • Interferon-gamma Release Tests*
  • Interleukin-2 / metabolism
  • Latent Tuberculosis / diagnosis*
  • Male
  • Middle Aged
  • Tumor Necrosis Factor-alpha / metabolism

Substances

  • Adrenal Cortex Hormones
  • Antirheumatic Agents
  • Interleukin-2
  • Tumor Necrosis Factor-alpha
  • Dexamethasone
  • Interferon-gamma
  • Infliximab