Role of the CX3C chemokine receptor CX3CR1 in the pathogenesis of atherosclerosis after aortic transplantation

PLoS One. 2017 Feb 24;12(2):e0170644. doi: 10.1371/journal.pone.0170644. eCollection 2017.

Abstract

Background: The CX3C chemokine receptor CX3CR1 is expressed on monocytes as well as tissue resident cells, such as smooth muscle cells (SMCs). Its role in atherosclerotic tissue remodeling of the aorta after transplantation has not been investigated.

Methods: We here have orthotopically transplanted infrarenal Cx3cr1-/-Apoe-/- and Cx3cr1+/+Apoe-/- aortic segments into Apoe-/-mice, as well as Apoe-/- aortic segments into Cx3cr1-/-Apoe-/- mice. The intimal plaque size and cellular plaque composition of the transplanted aortic segment were analyzed after four weeks of atherogenic diet.

Results: Transplantation of Cx3cr-/-Apoe-/- aortic segments into Apoe-/- mice resulted in reduced atherosclerotic plaque formation compared to plaque size in Apoe-/- or Cx3cr1-/-Apoe-/- mice after transplantation of Apoe-/- aortas. This reduction in lesion formation was associated with reduced numbers of lesional SMCs but not macrophages within the transplanted Cx3cr-/- Apoe-/- aortic segment. No differences in frequencies of proliferating and apoptotic cells could be observed.

Conclusion: These results indicate that CX3CR1 on resident vessel wall cells plays a key role in atherosclerotic plaque formation in transplanted aortic grafts. Targeting of vascular CX3CL1/CX3CR1 may therefore be explored as a therapeutic option in vascular transplantation procedures.

MeSH terms

  • Animals
  • Aorta / physiopathology
  • Aorta / transplantation*
  • Apolipoproteins E / genetics*
  • Atherosclerosis / genetics
  • Atherosclerosis / physiopathology
  • Atherosclerosis / therapy*
  • CX3C Chemokine Receptor 1
  • Chemokines, CX3C
  • Disease Models, Animal
  • Humans
  • Macrophages
  • Mice
  • Mice, Knockout
  • Monocytes
  • Myocytes, Smooth Muscle / metabolism
  • Myocytes, Smooth Muscle / pathology
  • Plaque, Atherosclerotic / genetics
  • Plaque, Atherosclerotic / physiopathology
  • Plaque, Atherosclerotic / therapy
  • Receptors, Chemokine / genetics*
  • Tunica Intima / metabolism
  • Tunica Intima / physiopathology

Substances

  • Apolipoproteins E
  • CX3C Chemokine Receptor 1
  • Chemokines, CX3C
  • Cx3cr1 protein, mouse
  • Receptors, Chemokine

Grants and funding

This study was supported by the Interdisciplinary Centre for Clinical Research IZKF Aachen (junior research group to E.L.) within the faculty of Medicine at RWTH Aachen University.