LKY-047: First Selective Inhibitor of Cytochrome P450 2J2

Drug Metab Dispos. 2017 Jul;45(7):765-769. doi: 10.1124/dmd.117.075036. Epub 2017 May 1.

Abstract

Highly selective cytochrome P450 CYP2J2 (CYP2J2) inhibitors suitable for reaction phenotyping are currently not available. (7S)-(+)-(4-Nitro-phenyl)-acrylic acid, 8,8-dimethyl-2-oxo-6,7-dihydro-2H,8H-pyrano[3,2-g]chromen-7-yl-ester (LKY-047), a decursin derivative, was synthesized, and its inhibitor potencies toward CYP2J2 as well as other cytochrome P450 (P450) enzymes in human liver microsomes (HLM) were evaluated. LKY-047 was demonstrated to be a strong competitive inhibitor of CYP2J2-mediated astemizole O-demethylase and terfenadine hydroxylase activity, with Ki values of 0.96 and 2.61 μM, respectively. It also acted as an uncompetitive inhibitor of CYP2J2-mediated ebastine hydroxylation with a Ki value of 3.61 μM. Preincubation of LKY-047 with HLMs and NADPH did not alter inhibition potency, indicating that it is not a mechanism-based inhibitor. LKY-047 was found to be a selective CYP2J2 inhibitor with no inhibitory effect on other human P450s, such as CYPs 1A2, 2A6, 2B6, 2C8, 2C9, 2C19, 2D6, 2E1, and 3A (IC50 > 50 μM). These in vitro data support the use of LKY-047 as a selective CYP2J2 inhibitor with potential application in the identification of P450 isoforms responsible for drug metabolism in reaction phenotyping assays.

MeSH terms

  • Cytochrome P-450 CYP2J2
  • Cytochrome P-450 Enzyme Inhibitors / pharmacology*
  • Cytochrome P-450 Enzyme System / metabolism*
  • Humans
  • Hydroxylation / drug effects
  • Inactivation, Metabolic / drug effects
  • Microsomes, Liver / drug effects
  • Microsomes, Liver / metabolism
  • NADP / metabolism
  • Protein Isoforms / metabolism

Substances

  • CYP2J2 protein, human
  • Cytochrome P-450 Enzyme Inhibitors
  • Protein Isoforms
  • NADP
  • Cytochrome P-450 Enzyme System
  • Cytochrome P-450 CYP2J2