A single amino acid change in the non-structural NV protein impacts the virulence phenotype of Viral hemorrhagic septicemia virus in trout

J Gen Virol. 2017 Jun;98(6):1181-1184. doi: 10.1099/jgv.0.000830. Epub 2017 Jun 22.

Abstract

Novirhabdoviruses like the Viral hemorrhagic septicemia virus (VHSV) are rhabdoviruses infecting fish. In the current study, RNA genomes of different VHSV field isolates classified as high, medium or low virulent phenotypes have been sequenced by next-generation sequencing and compared. Various amino acid changes, depending on the VHSV phenotype, have been identified in all the VHSV proteins. As a starting point, we focused our study on the non-virion (NV) non-structural protein in which an arginine residue (R116) is present in all the virulent isolates and replaced by a serine/asparagine residue S/N116 in the attenuated isolates. A recombinant virus derived from a virulent VHSV strain in which the NV R116 residue has been replaced by a serine, rVHSVNVR116S, was generated by reverse genetics and used to infect juvenile trout. We showed that rVHSVNVR116S was highly attenuated and that surviving fish were almost completely protected from a challenge with the wild-type VHSV.

MeSH terms

  • Amino Acid Substitution*
  • Animals
  • Fish Diseases / pathology*
  • Fish Diseases / virology*
  • Genome, Viral
  • Novirhabdovirus / genetics
  • Novirhabdovirus / isolation & purification
  • Novirhabdovirus / pathogenicity*
  • Phenotype
  • Reverse Genetics
  • Rhabdoviridae Infections / pathology
  • Rhabdoviridae Infections / veterinary*
  • Rhabdoviridae Infections / virology
  • Sequence Analysis, DNA
  • Trout
  • Viral Proteins / genetics*
  • Virulence
  • Virulence Factors / genetics*

Substances

  • NV protein, Rhabdovirus
  • Viral Proteins
  • Virulence Factors