Doxorubicin-loaded redox-responsive micelles based on dextran and indomethacin for resistant breast cancer

Int J Nanomedicine. 2017 Aug 22:12:6153-6168. doi: 10.2147/IJN.S141229. eCollection 2017.

Abstract

Multidrug resistance (MDR) against chemotherapeutic agents has become one of the major obstacles to successful cancer therapy and MDR-associated proteins (MRPs)-mediated drug efflux is the key factor for MDR. In this study, a redox-responsive polymer based on dextran (DEX) and indomethacin (IND), which could reduce MRPs-mediated efflux of chemotherapeutics, was synthesized, and the obtained polymer could spontaneously form stable micelles with well-defined core-shell structure and a uniform size distribution with an average diameter of 50 nm and effectively encapsulate doxorubicin (DOX); the micelles contain a disulfide bridge (cystamine, SS) between IND and DEX (DEX-SS-IND). In vitro drug release results indicated that DEX-SS-IND/DOX micelles could maintain good stability in a stimulated normal physiological environment and promptly depolymerized and released DOX in a reducing environment. After incubating DEX-SS-IND/DOX micelles with drug-resistant tumor (MCF-7/ADR) cells, the intracellular accumulation and retention of DOX were significantly increased under the synergistic effects of redox-responsive delivery and the inhibitory effect of IND on MRPs. In vitro cytotoxicity showed that DEX-SS-IND/DOX micelles exhibited higher cytotoxicity against MCF-7/ADR cells. Moreover, DEX-SS-IND/DOX micelles showed significantly enhanced inhibition of tumor in BALB/c nude mice bearing MCF-7/ADR tumors and reduced systemic toxicity. Overall, the cumulative evidence indicates that DEX-SS-IND/DOX micelles hold significant promise for overcoming MDR for cancer therapy.

Keywords: breast cancer; doxorubicin; indomethacin; micelles; multidrug resistance; redox-responsive.

MeSH terms

  • Animals
  • Antineoplastic Agents / administration & dosage*
  • Antineoplastic Agents / chemistry
  • Antineoplastic Agents / pharmacokinetics
  • Breast Neoplasms / drug therapy*
  • Dextrans / administration & dosage
  • Dextrans / chemistry
  • Doxorubicin / administration & dosage*
  • Doxorubicin / chemistry
  • Doxorubicin / pharmacokinetics
  • Drug Delivery Systems / methods*
  • Drug Liberation
  • Drug Resistance, Neoplasm / drug effects*
  • Female
  • Humans
  • Indomethacin / administration & dosage
  • Indomethacin / chemistry
  • MCF-7 Cells
  • Male
  • Mice, Inbred BALB C
  • Mice, Nude
  • Micelles
  • Oxidation-Reduction
  • Rats, Sprague-Dawley

Substances

  • Antineoplastic Agents
  • Dextrans
  • Micelles
  • Doxorubicin
  • Indomethacin