Effect of ethanol on cardiac beta-adrenoceptors

Eur J Pharmacol. 1987 Oct 27;142(3):441-5. doi: 10.1016/0014-2999(87)90085-9.

Abstract

Liquid diets are commonly used as vehicles for chronic administration of ethanol to rodents. After mice had consumed an ethanol-free liquid diet for either seven or eight days, the number of cardiac beta-adrenoceptors and the maximum response of adenylate cyclase to isoproterenol were decreased. This change was associated with a decrease in the number of high-affinity agonist binding sites. When mice were fed ethanol in the liquid diet, there was a further decrease in the number of cardiac low-affinity agonist (isoproterenol) binding sites, but no further change in the biochemical response to isoproterenol. The data suggest that stress and/or nutritional factors can alter the number, the coupling and the function of cardiac beta-adrenoceptors and that chronic ethanol ingestion enhances certain aspects of these changes.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adenylyl Cyclases / metabolism
  • Adrenergic beta-Agonists / metabolism
  • Adrenergic beta-Agonists / pharmacology
  • Adrenergic beta-Antagonists / metabolism
  • Adrenergic beta-Antagonists / pharmacology
  • Animals
  • Diet
  • Ethanol / pharmacology*
  • Iodine Radioisotopes
  • Isoproterenol / pharmacology
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Myocardium / enzymology
  • Myocardium / metabolism*
  • Receptors, Adrenergic, beta / drug effects
  • Receptors, Adrenergic, beta / metabolism*

Substances

  • Adrenergic beta-Agonists
  • Adrenergic beta-Antagonists
  • Iodine Radioisotopes
  • Receptors, Adrenergic, beta
  • Ethanol
  • Adenylyl Cyclases
  • Isoproterenol