Plasma cell survival in the absence of B cell memory

Nat Commun. 2017 Nov 24;8(1):1781. doi: 10.1038/s41467-017-01901-w.

Abstract

Pre-existing serum antibodies play an important role in vaccine-mediated protection against infection but the underlying mechanisms of immune memory are unclear. Clinical studies indicate that antigen-specific antibody responses can be maintained for many years, leading to theories that reactivation/differentiation of memory B cells into plasma cells is required to sustain long-term antibody production. Here, we present a decade-long study in which we demonstrate site-specific survival of bone marrow-derived plasma cells and durable antibody responses to multiple virus and vaccine antigens in rhesus macaques for years after sustained memory B cell depletion. Moreover, BrdU+ cells with plasma cell morphology can be detected for 10 years after vaccination/BrdU administration, indicating that plasma cells may persist for a prolonged period of time in the absence of cell division. On the basis of these results, long-lived plasma cells represent a key cell population responsible for long-term antibody production and serological memory.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antibodies, Bacterial / immunology
  • Antibody Formation
  • B-Lymphocytes / immunology*
  • Bacterial Vaccines / administration & dosage
  • Bacterial Vaccines / immunology
  • Cell Survival
  • Immunologic Memory*
  • Macaca mulatta
  • Male
  • Plasma Cells / cytology
  • Plasma Cells / immunology*
  • Vaccination

Substances

  • Antibodies, Bacterial
  • Bacterial Vaccines