MGMT pyrosequencing-based cut-off methylation level and clinical outcome in patients with glioblastoma multiforme

Future Oncol. 2018 Apr;14(8):699-707. doi: 10.2217/fon-2017-0437. Epub 2018 Mar 9.

Abstract

Aim: MGMT promoter methylation has been associated with improved survival in glioblastoma multiforme treated with temozolomide. However, there is no consensus on specific cut-off levels of methylation. The aims of the study were to explore the prognostic impact of MGMT methylation status and to analyze the role of specific cut-off values.

Materials & methods: We analyzed 108 glioblastoma multiforme patients treated between 2008 and 2013 stratified according to three pyrosequencing-based quantitative methylation in: unmethylated (methylation <9%), intermediate (9-29%) and highly methylated (>29%).

Results: The three-class stratification has a prognostic impact (median progression-free survival: 7.97, 11.6 and 15 months respectively; p = 0.004; median OS: 13.2, 15.8 and 19.5 months, respectively; p = 0.0002), especially in patients exposed to temozolomide.

Conclusion: Our study confirmed that the independent prognostic role of MGMT methylation status. An average level of methylation between all investigated CpGs of 9% may help discriminating between methylated and unmethylated tumors.

Keywords: CpG islands; IDH1; MGMT; cut-off; glioblastoma; methylation; pyrosequencing; temozolomide.

MeSH terms

  • Aged
  • CpG Islands / genetics
  • DNA Methylation / drug effects*
  • DNA Modification Methylases / genetics*
  • DNA Repair Enzymes / genetics*
  • Dacarbazine / administration & dosage
  • Dacarbazine / analogs & derivatives
  • Disease-Free Survival
  • Female
  • Glioblastoma / diet therapy*
  • Glioblastoma / genetics
  • Glioblastoma / pathology
  • Humans
  • Kaplan-Meier Estimate
  • Male
  • Middle Aged
  • Prognosis*
  • Promoter Regions, Genetic / drug effects
  • Temozolomide
  • Tumor Suppressor Proteins / genetics*

Substances

  • Tumor Suppressor Proteins
  • Dacarbazine
  • DNA Modification Methylases
  • MGMT protein, human
  • DNA Repair Enzymes
  • Temozolomide