Numb-p72, but not Numb-p65, contributes to the trafficking of group I metabotropic glutamate receptors

Neuroreport. 2018 Aug 1;29(11):902-906. doi: 10.1097/WNR.0000000000001051.

Abstract

The protein Numb localizes to clathrin-coated vesicles and participates in the trafficking of transmembrane receptors. We previous reported that Numb promotes the presence of metabotropic glutamate receptor 1 (mGlu1) on neuronal membrane, and Numb deficiency impairs synaptic expression of mGlu1. However, the actions of different Numb isoforms on mGlu1 trafficking are unknown. Here, we found that Numb-p72 and Numb-p65 are distinctly expressed in HEK293T cells. Interestingly, Numb-p72, but not Numb-p65, binds to mGlu1α and promotes the membrane expression of mGlu1α by antagonizing its internalization. We hypothesize that the incomplete structure of Numb-p65 does not act in the same way as Numb-p72 on mGlu1 trafficking.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cell Movement / physiology*
  • HEK293 Cells
  • Humans
  • Membrane Proteins / metabolism*
  • Nerve Tissue Proteins / metabolism*
  • Neurons / metabolism
  • Protein Transport / physiology*
  • Receptors, Metabotropic Glutamate / metabolism*

Substances

  • Membrane Proteins
  • Nerve Tissue Proteins
  • NUMB protein, human
  • Receptors, Metabotropic Glutamate
  • metabotropic glutamate receptor type 1