Simultaneous involvement of 11q23 translocation resulting in chimeric MLL-AFF1 and a second translocation [t (9;21) (p13; p11.2)] in an infant acute lymphoblastic leukemia patient at relapse: A case report

Medicine (Baltimore). 2018 May;97(21):e10874. doi: 10.1097/MD.0000000000010874.

Abstract

Rationale: Three-way translocations occasionally occur in MLL-AFF1 fusion and other fusion gene. However, the complex chromosomal rearrangements in the study were the first report.

Patient concerns: We present novel cryptic and complex chromosomal rearrangements [der (21) t (9; 21) (p13; p11.2)] in an infant patient with relapsed acute lymphoblastic leukemia (ALL).

Diagnoses: The diagnosis was based on morphologic, cytochemical, and immunophenotypic criteria proposed by the French-American-British Committee, and karyotype, fluorescence in situ hybridization, array comparative genomic hybridization.

Interventions: The patient was given chemotherapy with standard protocol for ALL.

Outcomes: The patient had unfavorable prognostic outcome based on the cytogenetic and molecular cytogenetic markers. After short remission, the patient relapsed.

Lessons: MLL-AFF1, resulting from t(4;11)(q21;q23), is regarded as the hallmark of infant t(4;11) pre-B/mixed B-ALL. It is associated with a dismal prognosis and the multiple-way translocation involving chromosomes 4, 11 and 11 may function as an enhancer.

Publication types

  • Case Reports

MeSH terms

  • Chromosome Aberrations
  • Chromosomes, Human, Pair 11 / genetics
  • Chromosomes, Human, Pair 4 / genetics
  • Comparative Genomic Hybridization / methods
  • Cytogenetic Analysis / methods
  • DNA-Binding Proteins / genetics*
  • Gene Fusion
  • Histone-Lysine N-Methyltransferase / genetics
  • Humans
  • Immunophenotyping
  • In Situ Hybridization, Fluorescence / methods*
  • Induction Chemotherapy / methods
  • Infant
  • Karyotyping
  • Male
  • Myeloid-Lymphoid Leukemia Protein / genetics
  • Precursor Cell Lymphoblastic Leukemia-Lymphoma / drug therapy
  • Precursor Cell Lymphoblastic Leukemia-Lymphoma / genetics*
  • Recurrence
  • Transcriptional Elongation Factors / genetics*
  • Translocation, Genetic / genetics*

Substances

  • DNA-Binding Proteins
  • KMT2A protein, human
  • Transcriptional Elongation Factors
  • Myeloid-Lymphoid Leukemia Protein
  • AFF1 protein, human
  • Histone-Lysine N-Methyltransferase