Human Clinical-Grade Parthenogenetic ESC-Derived Dopaminergic Neurons Recover Locomotive Defects of Nonhuman Primate Models of Parkinson's Disease

Stem Cell Reports. 2018 Jul 10;11(1):171-182. doi: 10.1016/j.stemcr.2018.05.010. Epub 2018 Jun 14.

Abstract

Clinical application of stem cell derivatives requires clinical-grade cells and sufficient preclinical proof of safety and efficacy, preferably in primates. We previously successfully established a clinical-grade human parthenogenetic embryonic stem cell (hPESC) line, but the suitability of its subtype-specific progenies for therapy is not clear. Here, we compared the function of clinical-grade hPESC-derived midbrain dopaminergic (DA) neurons in two canonical protocols in a primate Parkinson's disease (PD) model. We found that the grafts did not form tumors and produced variable but apparent behavioral improvement for at least 24 months in most monkeys in both groups. In addition, a slight DA increase in the striatum correlates with significant functional improvement. These results demonstrated that clinical-grade hPESCs can serve as a reliable source of cells for PD treatment. Our proof-of-concept findings provide preclinical data for China's first ESC-based phase I/IIa clinical study of PD (ClinicalTrials.gov number NCT03119636).

Keywords: Parkinson's disease; cell therapy; dopaminergic neuron; embryonic stem cell; monkey; nonhuman primate.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Behavior, Animal
  • Biomarkers
  • Brain / cytology
  • Brain / metabolism
  • Cell Differentiation
  • Cell Line
  • Cell Movement
  • Cell Survival
  • Cell Transformation, Neoplastic
  • Cell- and Tissue-Based Therapy
  • Disease Models, Animal
  • Dopamine / metabolism
  • Dopaminergic Neurons / cytology*
  • Dopaminergic Neurons / physiology*
  • Embryonic Stem Cells / cytology*
  • Humans
  • Locomotion*
  • Neural Stem Cells / cytology
  • Neural Stem Cells / metabolism
  • Parkinson Disease / etiology
  • Parkinson Disease / physiopathology*
  • Parkinson Disease / therapy*
  • Phenotype
  • Primates
  • Putamen / metabolism
  • Putamen / physiopathology

Substances

  • Biomarkers
  • Dopamine

Associated data

  • ClinicalTrials.gov/NCT03119636