Abstract
LR and [d-Gln4]LR peptides bind the monomer-monomer interface of human thymidylate synthase and inhibit cancer cell growth. Here, proline-mutated LR peptides were synthesized. Molecular dynamics calculations and circular dichroism spectra have provided a consistent picture of the conformational propensities of the [Pro n]-peptides. [Pro3]LR and [Pro4]LR show improved cell growth inhibition and similar intracellular protein modulation compared with LR. These represent a step forward to the identification of more rigid and metabolically stable peptides.
Publication types
-
Research Support, Non-U.S. Gov't
MeSH terms
-
Antineoplastic Agents / chemistry
-
Antineoplastic Agents / pharmacology*
-
Cell Line, Tumor
-
Circular Dichroism
-
Enzyme Inhibitors / chemistry
-
Enzyme Inhibitors / pharmacology*
-
Female
-
Humans
-
Molecular Dynamics Simulation
-
Mutation
-
Ovarian Neoplasms / drug therapy*
-
Ovarian Neoplasms / pathology
-
Peptides / chemistry
-
Peptides / genetics
-
Peptides / pharmacology*
-
Proline / genetics
-
Protein Conformation
-
Thymidylate Synthase / antagonists & inhibitors*
-
Thymidylate Synthase / genetics
-
Thymidylate Synthase / metabolism
Substances
-
Antineoplastic Agents
-
Enzyme Inhibitors
-
Peptides
-
Proline
-
Thymidylate Synthase